Imp2 controls oxidative phosphorylation and is crucial for preserving glioblastoma cancer stem cells. - Wikidata - thesis-toulmin - TriplyDB

Imp2 controls oxidative phosphorylation and is crucial for preserving glioblastoma cancer stem cells.

Imp2 controls oxidative phosphorylation and is crucial for preserving glioblastoma cancer stem cells.

http://www.wikidata.org/entity/Q36213711

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SVD identifies transcript length distribution functions from DNA microarray data and reveals evolutionary forces globally affecting GBM metabolism Cancer stem cell metabolism Lin28 and let-7 in cell metabolism and cancer Bypassing Mechanisms of Mitochondria-Mediated Cancer Stem Cells Resistance to Chemo- and Radiotherapy Harnessing the apoptotic programs in cancer stem-like cells Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance.PI3K therapy reprograms mitochondrial trafficking to fuel tumor cell invasion.Survivin promotes oxidative phosphorylation, subcellular mitochondrial repositioning, and tumor cell invasion Cancer stem cell metabolism: a potential target for cancer therapy Lin28: primal regulator of growth and metabolism in stem cells A combinatorial radiographic phenotype may stratify patient survival and be associated with invasion and proliferation characteristics in glioblastoma Mitochondrial control by DRP1 in brain tumor initiating cells.Selective Vulnerability of Cancer Cells by Inhibition of Ca(2+) Transfer from Endoplasmic Reticulum to Mitochondria Coordinated metabolic transitions during Drosophila embryogenesis and the onset of aerobic glycolysis.Head and Neck Squamous Cell Carcinoma Metabolism Draws on Glutaminolysis, and Stemness Is Specifically Regulated by Glutaminolysis via Aldehyde Dehydrogenase The evolving landscape of glioblastoma stem cells.Adenylate kinase hCINAP determines self-renewal of colorectal cancer stem cells by facilitating LDHA phosphorylation.Contribution of the Microenvironmental Niche to Glioblastoma Heterogeneity.Repositioning chlorpromazine for treating chemoresistant glioma through the inhibition of cytochrome c oxidase bearing the COX4-1 regulatory subunit.Deletion of the mitochondrial chaperone TRAP-1 uncovers global reprogramming of metabolic networks.TLR9 is critical for glioma stem cell maintenance and targeting.Identification and characterization of alternative exon usage linked glioblastoma multiforme survival Mitochondria as therapeutic targets for cancer stem cells.Fbxl10 overexpression in murine hematopoietic stem cells induces leukemia involving metabolic activation and upregulation of Nsg2.Knockdown of NAT12/NAA30 reduces tumorigenic features of glioblastoma-initiating cells Cancer Stem Cells in Small Cell Lung Cancer Cell Line H446: Higher Dependency on Oxidative Phosphorylation and Mitochondrial Substrate-Level Phosphorylation than Non-Stem Cancer Cells Mitochondrial-Targeted Decyl-Triphenylphosphonium Enhances 2-Deoxy-D-Glucose Mediated Oxidative Stress and Clonogenic Killing of Multiple Myeloma Cells Imp2 regulates GBM progression by activating IGF2/PI3K/Akt pathway.IGF2BP2/IMP2-Deficient mice resist obesity through enhanced translation of Ucp1 mRNA and Other mRNAs encoding mitochondrial proteins.Molecular Pathways: Mitochondrial Reprogramming in Tumor Progression and Therapy.Cell context-dependent dual effects of EFEMP1 stabilizes subpopulation equilibrium in responding to changes of in vivo growth environment p62/IMP2 stimulates cell migration and reduces cell adhesion in breast cancer.Active glycolytic metabolism in CD133(+) hepatocellular cancer stem cells: regulation by MIR-122.IMP3 protein promotes chemoresistance in breast cancer cells by regulating breast cancer resistance protein (ABCG2) expression.Enhanced CLIP Uncovers IMP Protein-RNA Targets in Human Pluripotent Stem Cells Important for Cell Adhesion and Survival The Long Non-coding RNA HIF1A-AS2 Facilitates the Maintenance of Mesenchymal Glioblastoma Stem-like Cells in Hypoxic Niches HIF-1-alpha links mitochondrial perturbation to the dynamic acquisition of breast cancer tumorigenicity.IMPs: an RNA-binding protein family that provides a link between stem cell maintenance in normal development and cancer.Elevated expression of the IGF2 mRNA binding protein 2 (IGF2BP2/IMP2) is linked to short survival and metastasis in esophageal adenocarcinoma.Heterogeneity in Cancer Metabolism: New Concepts in an Old Field.

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Imp2 controls oxidative phosphorylation and is crucial for preserving glioblastoma cancer stem cells.

http://www.wikidata.org/entity/Q36213711

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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name

Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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type

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

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Genes & Development

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Imp2 controls oxidative phosph ...... lioblastoma cancer stem cells.

@en

P2093

Esther Rheinbay

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Ivan Radovanovic

http://www.w3.org/2001/XMLSchema#string

Ivan Stamenkovic

http://www.w3.org/2001/XMLSchema#string

Mario L Suvà

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Michalina Janiszewska

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Nicolo Riggi

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Paolo Provero

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Riekelt H Houtkooper

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Virginie Clément-Schatlo

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P2860

A family of insulin-like growth factor II mRNA-binding proteins represses translation in late development

Hallmarks of Cancer: The Next Generation

The subunit composition of the human NADH dehydrogenase obtained by rapid one-step immunopurification

Understanding the Warburg effect: the metabolic requirements of cell proliferation

mTOR: from growth signal integration to cancer, diabetes and ageing

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

Identification and expansion of human colon-cancer-initiating cells

Identification of human brain tumour initiating cells

A cell initiating human acute myeloid leukaemia after transplantation into SCID mice

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1926-1944

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scholarly article

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10.1101/GAD.188292.112

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17

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26

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2012-08-16T00:00:00Z

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230685568

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22899010

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phosphorylation

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3435496

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