Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
about
Functional interactions among members of the MAX and MLX transcriptional network during oncogenesisInduction of a novel histone deacetylase 1/c-Myc/Mnt/Max complex formation is implicated in parity-induced refractoriness to mammary carcinogenesisNucleophosmin suppresses oncogene-induced apoptosis and senescence and enhances oncogenic cooperation in cells with genomic instability.The transcriptional repressor dMnt is a regulator of growth in Drosophila melanogasterIkaros and Aiolos inhibit pre-B-cell proliferation by directly suppressing c-Myc expressionA critical role for Mnt in Myc-driven T-cell proliferation and oncogenesisInflammatory disease and lymphomagenesis caused by deletion of the Myc antagonist Mnt in T cells.Identification of small molecules that induce apoptosis in a Myc-dependent manner and inhibit Myc-driven transformationProteomic discovery of Max as a novel interacting partner of C/EBPalpha: a Myc/Max/Mad link.Initial testing (stage 1) of the polyamine analog PG11047 by the pediatric preclinical testing program.Defining the role of polyamines in colon carcinogenesis using mouse models.Evolution of the Max and Mlx networks in animals.Myc targets Cks1 to provoke the suppression of p27Kip1, proliferation and lymphomagenesis.Proteomic discovery of MNT as a novel interacting partner of E3 ubiquitin ligase E6AP and a key mediator of myeloid differentiation.Mnt-Max to Myc-Max complex switching regulates cell cycle entry.Drosophila growth and development in the absence of dMyc and dMnt.OX40 engagement stabilizes Mxd4 and Mnt protein levels in antigen-stimulated T cells leading to an increase in cell survival.Current status of the polyamine research fieldMyc's broad reach.Mammalian polyamine metabolism and functionAn overview of MYC and its interactome.MiR-146b-5p overexpression attenuates stemness and radioresistance of glioma stem cells by targeting HuR/lincRNA-p21/β-catenin pathway.The centella asiatica juice effects on DNA damage, apoptosis and gene expression in hepatocellular carcinoma (HCC).MNT and Emerging Concepts of MNT-MYC AntagonismThe direct Myc target Pim3 cooperates with other Pim kinases in supporting viability of Myc-induced B-cell lymphomas.Switch from Mnt-Max to Myc-Max induces p53 and cyclin D1 expression and apoptosis during cholestasis in mouse and human hepatocytes.Polyamine analogues: potent inducers of nucleosomal array oligomerization and inhibitors of yeast cell growth.Max-independent functions of Myc in Drosophila melanogaster.Mnt modulates Myc-driven lymphomagenesis.Pathogenesis and therapeutic targeting of aberrant MYC expression in haematological cancers.Pan-cancer Alterations of the MYC Oncogene and Its Proximal Network across the Cancer Genome Atlas.
P2860
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P2860
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@ast
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@en
type
label
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@ast
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@en
prefLabel
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@ast
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@en
P2093
P2860
P1476
Mnt loss triggers Myc transcription targets, proliferation, apoptosis, and transformation
@en
P2093
Helene Pendeville
John L Cleveland
Jonas A Nilsson
Kirsteen H Maclean
Troy A Baudino
Ulrich B Keller
P2860
P304
P356
10.1128/MCB.24.4.1560-1569.2004
P407
P577
2004-02-01T00:00:00Z