Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.
about
Neuroimmunological Implications of AQP4 in AstrocytesAquaporins in the Spinal CordMechanisms of Autoantibody-Induced Pathology.Experimental Neuromyelitis Optica Induces a Type I Interferon Signature in the Spinal Cord.A Single Amino Acid Substitution Prevents Recognition of a Dominant Human Aquaporin-4 Determinant in the Context of HLA-DRB1*03:01 by a Murine TCR.Lack of KIR4.1 autoantibodies in Japanese patients with MS and NMO.Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SDTolerance checkpoint bypass permits emergence of pathogenic T cells to neuromyelitis optica autoantigen aquaporin-4.Multiple sclerosis: experimental models and reality.Upregulation of Bcl-2 and Its Promoter Signals in CD4+ T Cells during Neuromyelitis Optica Remission.Marked central nervous system pathology in CD59 knockout rats following passive transfer of Neuromyelitis optica immunoglobulin G.Deletional tolerance prevents AQP4-directed autoimmunity in mice.Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder.T cells targeting neuromyelitis optica autoantigen aquaporin-4 cause paralysis and visual system injury.The Role of Brain-Reactive Autoantibodies in Brain Pathology and Cognitive Impairment.Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4.Selective Limbic Blood-Brain Barrier Breakdown in a Feline Model of Limbic Encephalitis with LGI1 Antibodies.Association of TNFSF4 Polymorphisms with Neuromyelitis Optica Spectrum Disorders in a Chinese Population.Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Early and extensive spinal white matter involvement in neuromyelitis optica.
P2860
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P2860
Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@ast
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@en
type
label
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@ast
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@en
prefLabel
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@ast
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@en
P2093
P2860
P50
P1476
Highly encephalitogenic aquapo ...... and tissue damage in the CNS.
@en
P2093
Bleranda Zeka
Fritz Leutmezer
Ichiro Nakashima
Kathrin Schanda
Kazuo Fujihara
Maria Hastermann
Nathalie Kaufmann
Nikolaus Kögl
Paulus Rommer
Simone Mader
P2860
P2888
P304
P356
10.1007/S00401-015-1501-5
P577
2015-11-03T00:00:00Z