SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
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Novel endogenous angiogenesis inhibitors and their therapeutic potentialSOCS3: An essential physiological inhibitor of signaling by interleukin-6 and G-CSF family cytokinesThe future of EPAC-targeted therapies: agonism versus antagonismIL-10 inhibits the NF-κB and ERK/MAPK-mediated production of pro-inflammatory mediators by up-regulation of SOCS-3 in Trypanosoma cruzi-infected cardiomyocytes.Sirtuin1 over-expression does not impact retinal vascular and neuronal degeneration in a mouse model of oxygen-induced retinopathy.Dietary ω-3 polyunsaturated fatty acids decrease retinal neovascularization by adipose-endoplasmic reticulum stress reduction to increase adiponectin.Endothelial TWIST1 promotes pathological ocular angiogenesis.Nuclear receptor RORα regulates pathologic retinal angiogenesis by modulating SOCS3-dependent inflammation.SOCS3 in retinal neurons and glial cells suppresses VEGF signaling to prevent pathological neovascular growthIL10-driven STAT3 signalling in senescent macrophages promotes pathological eye angiogenesis.Effects of Controlled Cortical Impact on the Mouse Brain Vasculome.CNTF Attenuates Vasoproliferative Changes Through Upregulation of SOCS3 in a Mouse-Model of Oxygen-Induced Retinopathy.IDO1 is an Integral Mediator of Inflammatory Neovascularization.STAT3 inhibitors: finding a home in lymphoma and leukemia.Epigenetic approach for angiostatic therapy: promising combinations for cancer treatment.In vitro significance of SOCS-3 and SOCS-4 and potential mechanistic links to wound healing.Induction of SOCS3 by liver X receptor suppresses the proliferation of hepatocellular carcinoma cellsSOCS3-mediated blockade reveals major contribution of JAK2/STAT5 signaling pathway to lactation and proliferation of dairy cow mammary epithelial cells in vitro.Dysregulation of Calpain Proteolytic Systems Underlies Degenerative Vascular Disorders.Refuting the hypothesis that semaphorin-3f/neuropilin-2 exclude blood vessels from the cap mesenchyme in the developing kidney.The Potential of a Novel Class of EPAC-Selective Agonists to Combat Cardiovascular Inflammation.Time-dependent LXR/RXR pathway modulation characterizes capillary remodeling in inflammatory corneal neovascularization.Indoleamine 2,3-Dioxygenase and Its Therapeutic Inhibition in Cancer.Inflammatory Reprogramming with IDO1 Inhibitors: Turning Immunologically Unresponsive 'Cold' Tumors 'Hot'.Resistin causes G1 arrest in colon cancer cells through upregulation of SOCS3.Enhanced SOCS3 in osteoarthiritis may limit both proliferation and inflammation.
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P2860
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
@ast
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
@en
type
label
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
@ast
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
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prefLabel
SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
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SOCS3 is an endogenous inhibitor of pathologic angiogenesis.
@en
P2093
P2860
P1433
P1476
SOCS3 is an endogenous inhibitor of pathologic angiogenesis
@en
P2093
Andreas Stahl
Christian G Hurst
Colman J Hatton
Dipak Panigrahy
Dorothy T Pei
Emily R Greene
Jean-Sebastian Joyal
Lois E H Smith
Molly R Seaward
P2860
P304
P356
10.1182/BLOOD-2012-04-422527
P407
P577
2012-07-12T00:00:00Z