Consequences of the electrogenic function of the phagocytic NADPH oxidase. - Wikidata - thesis-toulmin - TriplyDB

Consequences of the electrogenic function of the phagocytic NADPH oxidase.

Consequences of the electrogenic function of the phagocytic NADPH oxidase.

http://www.wikidata.org/entity/Q36328364

about

Neutrophils in innate host defense against Staphylococcus aureus infections Small-molecule screen identifies reactive oxygen species as key regulators of neutrophil chemotaxis.VSOP/Hv1 proton channels sustain calcium entry, neutrophil migration, and superoxide production by limiting cell depolarization and acidification.Dysregulated Ca2+ homeostasis in Fanconi anemia cells.Primary involvement of NADPH oxidase 4 in hypoxia-induced generation of reactive oxygen species in adipose-derived stem cells The antibacterial activity of human neutrophils and eosinophils requires proton channels but not BK channels Hv1 proton channels are required for high-level NADPH oxidase-dependent superoxide production during the phagocyte respiratory burst Interactions between Neutrophils and Pseudomonas aeruginosa in Cystic Fibrosis.Diverse novel functions of neutrophils in immunity, inflammation, and beyond.Intracellular shunting of O2(-) contributes to charge compensation and preservation of neutrophil respiratory burst in the absence of voltage-gated proton channel activity.Charge compensation for NADPH oxidase activity in microglia in rat brain slices does not involve a proton current.Introduction: reactive oxygen species in health and disease.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

http://www.wikidata.org/entity/Q36328364

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Consequences of the electrogenic function of the phagocytic NADPH oxidase.

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Philosophical Transactions of the Royal Society B

P1476

Consequences of the electrogenic function of the phagocytic NADPH oxidase.

@en

P2093

Balázs K Rada

http://www.w3.org/2001/XMLSchema#string

Csilla Hably

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Erzsébet Ligeti

http://www.w3.org/2001/XMLSchema#string

Miklós Geiszt

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P2860

Killing activity of neutrophils is mediated through activation of proteases by K+ flux

New disguises for an old channel: MaxiK channel beta-subunits

Accelerated calcium influx and hyperactivation of neutrophils in chronic granulomatous disease

Cytosolic free Ca(2+) changes and calpain activation are required for beta integrin-accelerated phagocytosis by human neutrophils.

Voltage-gated proton channels and other proton transfer pathways.

Chronic granulomatous disease: more than the lack of superoxide?

The large-conductance Ca2+-activated K+ channel is essential for innate immunity.

Proton conduction through gp91phox.

Voltage-gated proton "channels": a spectator's viewpoint

The gp91phox component of NADPH oxidase is not a voltage-gated proton channel.

P304

2293-2300

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scholarly article

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10.1098/RSTB.2005.1768

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1464

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360

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2005-12-01T00:00:00Z

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7446878

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