Myeloperoxidase and plasminogen activator inhibitor 1 play a central role in ventricular remodeling after myocardial infarction.
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Cathepsin g is required for sustained inflammation and tissue injury after reperfusion of ischemic kidneysIncreased systemic and myocardial expression of neutrophil gelatinase-associated lipocalin in clinical and experimental heart failurePlasminogen activator inhibitor-1 (PAI-1) is cardioprotective in mice by maintaining microvascular integrity and cardiac architecture.Elevated plasma free fatty acids increase cardiovascular risk by inducing plasma biomarkers of endothelial activation, myeloperoxidase and PAI-1 in healthy subjectsPleiotropic functions of plasminogen activator inhibitor-1.Olmesartan prevents cardiac rupture in mice with myocardial infarction by modulating growth differentiation factor 15 and p53Association between peak neutrophil count, clopidogrel loading dose, and left ventricular systolic function in patients with primary percutaneous coronary intervention.Role of vegetation-associated protease activity in valve destruction in human infective endocarditis.Comprehensive peroxidase-based hematologic profiling for the prediction of 1-year myocardial infarction and death.Role of biomarkers in risk stratification of acute coronary syndromeIncreased expression of plasminogen activator inhibitor-1 in cardiomyocytes contributes to cardiac fibrosis after myocardial infarction.Differential regenerative capacity of neonatal mouse hearts after cryoinjury.Reactive oxygen species in cardiovascular disease.Matricellular proteins in cardiac adaptation and disease.Role of oxidative stress in disease progression in Stage B, a pre-cursor of heart failure.Early postmyocardial infarction survival in Murphy Roths Large mice is mediated by attenuated apoptosis and inflammation but depends on genetic backgroundDetection and quantification of fluorescent cell clusters in cryo-imaging.Fibrosis in Atrial Fibrillation - Role of Reactive Species and MPO.Arjunolic acid ameliorates reactive oxygen species via inhibition of p47(phox)-serine phosphorylation and mitochondrial dysfunctionPeriostin is essential for cardiac healing after acute myocardial infarction.Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activation.Identification of nondiabetic heart failure-associated genes by bioinformatics approaches in patients with dilated ischemic cardiomyopathy.Validation of the vitronectin knockout mouse as a model for studying myocardial infarction: Vitronectin appears to influence left ventricular remodelling following myocardial infarction.Atrial natriuretic peptide increases inflammation, infarct size, and mortality after experimental coronary occlusion.Genetic enhancement of stem cell engraftment, survival, and efficacy.Usefulness of myeloperoxidase levels in healthy elderly subjects to predict risk of developing heart failure.Potential novel pharmacological therapies for myocardial remodelling.Myeloperoxidase, modified lipoproteins, and atherogenesis.Modification of high density lipoprotein by myeloperoxidase generates a pro-inflammatory particle.Naoxintong attenuates Ischaemia/reperfusion Injury through inhibiting NLRP3 inflammasome activation.Evaluation of serum myeloperoxidase concentration in dogs with heart failure due to chronic mitral valvular insufficiency.Current role of myeloperoxidase in routine clinical practice.Risk stratification and short-term prognosis in acute heart failure syndromes: a review of novel biomarkers.Mitochondrial mitophagic mechanisms of myocardial matrix metabolism and remodelling.Neutrophil roles in left ventricular remodeling following myocardial infarction.Molecular imaging of macrophage enzyme activity in cardiac inflammation.Loss or inhibition of uPA or MMP-9 attenuates LV remodeling and dysfunction after acute pressure overload in mice.The Time Course of Markers of Neutrophil Extracellular Traps in Patients Undergoing Revascularisation for Acute Myocardial Infarction or Stable Angina Pectoris.Apolipoprotein A1 regulates coenzyme Q10 absorption, mitochondrial function, and infarct size in a mouse model of myocardial infarction.Myeloperoxidase is critically involved in the induction of organ damage after renal ischemia reperfusion.
P2860
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P2860
Myeloperoxidase and plasminogen activator inhibitor 1 play a central role in ventricular remodeling after myocardial infarction.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@ast
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@en
type
label
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@ast
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@en
prefLabel
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@ast
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@en
P2093
P2860
P356
P1476
Myeloperoxidase and plasminoge ...... g after myocardial infarction.
@en
P2093
Annitta Morehead
Arman T Askari
James D Thomas
Jeanne Drinko
Marc S Penn
Marie-Luise Brennan
Stanley L Hazen
Xiaorong Zhou
P2860
P304
P356
10.1084/JEM.20021426
P407
P50
P577
2003-03-01T00:00:00Z