The majority of dorsal spinal cord gastrin releasing peptide is synthesized locally whereas neuromedin B is highly expressed in pain- and itch-sensing somatosensory neurons - Wikidata - thesis-toulmin - TriplyDB

The majority of dorsal spinal cord gastrin releasing peptide is synthesized locally whereas neuromedin B is highly expressed in pain- and itch-sensing somatosensory neurons

The majority of dorsal spinal cord gastrin releasing peptide is synthesized locally whereas neuromedin B is highly expressed in pain- and itch-sensing somatosensory neurons

http://www.wikidata.org/entity/Q36391729

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Why we scratch an itch: the molecules, cells and circuits of itch New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Insights into bombesin receptors and ligands: Highlighting recent advances Neuromedin B and Its Receptor: Gene Cloning, Tissue Distribution and Expression Levels of the Reproductive Axis in Pigs Neural processing of itch.The cells and circuitry for itch responses in mice.Chronic itch development in sensory neurons requires BRAF signaling pathways.The peripheral and central mechanisms underlying itch Spinal cord interneurons expressing the gastrin-releasing peptide receptor convey itch through VGLUT2-mediated signaling Itch mechanisms and circuits.Itch-associated peptides: RNA-Seq and bioinformatic analysis of natriuretic precursor peptide B and gastrin releasing peptide in dorsal root and trigeminal ganglia, and the spinal cord.Cross-inhibition of NMBR and GRPR signaling maintains normal histaminergic itch transmission Role of spinal bombesin-responsive neurons in nonhistaminergic itch.Primary afferent and spinal cord expression of gastrin-releasing peptide: message, protein, and antibody concerns Expression of gastrin-releasing peptide by excitatory interneurons in the mouse superficial dorsal horn.Dual innervation of neonatal Merkel cells in mouse touch domes.Cis and trans RET signaling control the survival and central projection growth of rapidly adapting mechanoreceptors.Molecular and cellular mechanisms that initiate pain and itch.Gastrin-releasing peptide receptors in the central nervous system: role in brain function and as a drug target.Spinal Functions of B-Type Natriuretic Peptide, Gastrin-Releasing Peptide, and Their Cognate Receptors for Regulating Itch in Mice The TGR5 receptor mediates bile acid-induced itch and analgesia Spinal neurons that contain gastrin-releasing peptide seldom express Fos or phosphorylate extracellular signal-regulated kinases in response to intradermal chloroquine.A quantitative study of neurochemically defined excitatory interneuron populations in laminae I-III of the mouse spinal cord.Critical evaluation of the expression of gastrin-releasing peptide in dorsal root ganglia and spinal cord.Modality-based organization of ascending somatosensory axons in the direct dorsal column pathway B-type natriuretic peptide is neither itch-specific nor functions upstream of the GRP-GRPR signaling pathway Roles of glutamate, substance P, and gastrin-releasing peptide as spinal neurotransmitters of histaminergic and nonhistaminergic itch The Gastrin-Releasing Peptide Receptor (GRPR) in the Spinal Cord as a Novel Pharmacological Target The Stem Cell Marker Lgr5 Defines a Subset of Post-Mitotic Neurons in the Olfactory Bulb.Molecular dissection of itch.Transmission of pruriceptive signals.Spinal Mechanisms of Itch Transmission.Identifying functional populations among the interneurons in laminae I-III of the spinal dorsal horn.Comparative Anatomy of Gastrin-releasing Peptide Pathways in the Trigeminal Sensory System of Mouse and the Asian House Musk Shrew Suncus murinus.Sparse genetic tracing reveals regionally specific functional organization of mammalian nociceptors.TRPC3 Is Dispensable for β-Alanine Triggered Acute Itch.Gastrin-releasing peptide-expressing nerves comprise subsets of human cutaneous Aδ and C fibers that may sense pruritus.Spinal Circuits for Touch, Pain, and Itch.Distinct roles of NMB and GRP in itch transmission.Leaky Gate Model: Intensity-Dependent Coding of Pain and Itch in the Spinal Cord.

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The majority of dorsal spinal cord gastrin releasing peptide is synthesized locally whereas neuromedin B is highly expressed in pain- and itch-sensing somatosensory neurons

http://www.wikidata.org/entity/Q36391729

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The majority of dorsal spinal ...... -sensing somatosensory neurons

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Daniel Ramos

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Jianyuan Zhao

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Michael S Fleming

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Seung Baek Han

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Wenqin Luo

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Young-Jin Son

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P2860

Cellular basis of itch sensation

The capsaicin receptor: a heat-activated ion channel in the pain pathway

International Union of Pharmacology. LXVIII. Mammalian bombesin receptors: nomenclature, distribution, pharmacology, signaling, and functions in normal and disease states

Analyzing real-time PCR data by the comparative C(T) method

A diverse family of GPCRs expressed in specific subsets of nociceptive sensory neurons

Neuromedin B and gastrin-releasing peptide mRNAs are differentially distributed in the rat nervous system

A hierarchical NGF signaling cascade controls Ret-dependent and Ret-independent events during development of nonpeptidergic DRG neurons

TRPM8 is required for cold sensation in mice

TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.

VGLUT2-dependent sensory neurons in the TRPV1 population regulate pain and itch.

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