Gut motor function: immunological control in enteric infection and inflammation.
about
Upregulation of RGS4 expression by IL-1beta in colonic smooth muscle is enhanced by ERK1/2 and p38 MAPK and inhibited by the PI3K/Akt/GSK3beta pathwayGastrointestinal Parasites and the Neural Control of Gut FunctionsGut bacteria in health and diseaseDelayed goblet cell hyperplasia, acetylcholine receptor expression, and worm expulsion in SMC-specific IL-4Ralpha-deficient miceFood allergy alters jejunal circular muscle contractility and induces local inflammatory cytokine expression in a mouse model.Involvement of interleukin-17A-induced hypercontractility of intestinal smooth muscle cells in persistent gut motor dysfunctionAntioxidant activity of inulin and its role in the prevention of human colonic muscle cell impairment induced by lipopolysaccharide mucosal exposure.Hypercontractility of intestinal longitudinal smooth muscle induced by cytokines is mediated by the nuclear factor-κB/AMP-activated kinase/myosin light chain kinase pathway.Jun kinase-induced overexpression of leukemia-associated Rho GEF (LARG) mediates sustained hypercontraction of longitudinal smooth muscle in inflammation.Upregulation of RGS4 and downregulation of CPI-17 mediate inhibition of colonic muscle contraction by interleukin-1beta.Cytokine-induced iNOS and ERK1/2 inhibit adenylyl cyclase type 5/6 activity and stimulate phosphodiesterase 4D5 activity in intestinal longitudinal smooth muscle.What does irritable bowel syndrome share with non-alcoholic fatty liver disease?Low-grade inflammation plays a pivotal role in gastrointestinal dysfunction in irritable bowel syndrome.MAPKs represent novel therapeutic targets for gastrointestinal motility disorders.Cytokine-induced S-nitrosylation of soluble guanylyl cyclase and expression of phosphodiesterase 1A contribute to dysfunction of longitudinal smooth muscle relaxation.The effects of knockdown of rho-associated kinase 1 and zipper-interacting protein kinase on gene expression and function in cultured human arterial smooth muscle cells.Eosinophilic myenteric ganglionitis as a cause of chronic intestinal pseudo-obstructionCellular and Molecular Mechanisms of Phenotypic Switch in Gastrointestinal Smooth Muscle.Serum and colonic mucosal immune markers in irritable bowel syndromeImmunoinflammation and functional gastrointestinal disorders.Contribution of gender to pathophysiology and clinical presentation of IBS: should management be different in women?Immune/Inflammatory Response and Hypocontractility of Rabbit Colonic Smooth Muscle After TNBS-Induced Colitis.Trichuris muris research revisited: a journey through timePhysiological changes in the gastrointestinal tract and host protective immunity: learning from the mouse-Trichinella spiralis model.Changes in the expression of smooth muscle contractile proteins in TNBS- and DSS-induced colitis in mice.Brain-derived neurotrophic factor enhances cholinergic contraction of longitudinal muscle of rabbit intestine via activation of phospholipase CDrug discovery approaches to irritable bowel syndrome.Ageing and Gastrointestinal Sensory Function.Factors associated with clinical failure of self-expandable metal stent for malignant gastroduodenal obstruction.Activation of the silkworm cytokine by bacterial and fungal cell wall components via a reactive oxygen species-triggered mechanism.Schistosoma mansoni proteins attenuate gastrointestinal motility disturbances during experimental colitis in miceMicroflora modulation of motility.The study on the role of inflammatory cells and mediators in post-infectious functional dyspepsia.Carcinomatosis matters: clinical outcomes and prognostic factors for clinical success of stent placement in malignant gastric outlet obstruction.Chronic cigarette smoke exposure induces microbial and inflammatory shifts and mucin changes in the murine gut.Colonic motor dysfunctions in a mouse model of high-fat diet-induced obesity: an involvement of A2B adenosine receptors.Pathogenic mechanisms of postinfectious functional gastrointestinal disorders: results 3 years after gastroenteritis.The flavonoid compound apigenin prevents colonic inflammation and motor dysfunctions associated with high fat diet-induced obesity.The contribution of protein kinase C and CPI-17 signaling pathways to hypercontractility in murine experimental colitis.The DH31/CGRP enteroendocrine peptide triggers intestinal contractions favoring the elimination of opportunistic bacteria
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P2860
Gut motor function: immunological control in enteric infection and inflammation.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Gut motor function: immunological control in enteric infection and inflammation.
@ast
Gut motor function: immunological control in enteric infection and inflammation.
@en
type
label
Gut motor function: immunological control in enteric infection and inflammation.
@ast
Gut motor function: immunological control in enteric infection and inflammation.
@en
prefLabel
Gut motor function: immunological control in enteric infection and inflammation.
@ast
Gut motor function: immunological control in enteric infection and inflammation.
@en
P2860
P1476
Gut motor function: immunological control in enteric infection and inflammation.
@en
P2093
P2860
P304
P356
10.1111/J.1365-2249.2005.02979.X
P577
2006-03-01T00:00:00Z