FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions - Wikidata - thesis-toulmin - TriplyDB

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

http://www.wikidata.org/entity/Q36677983

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Functions and regulation of the multitasking FANCM family of DNA motor proteins Architecture and DNA Recognition Elements of the Fanconi Anemia FANCM-FAAP24 Complex Structure analysis of FAAP24 reveals single-stranded DNA-binding activity and domain functions in DNA damage response Structural insights into the functions of the FANCM-FAAP24 complex in DNA repair Forkhead transcription factor FoxF1 interacts with Fanconi anemia protein complexes to promote DNA damage response Advances in understanding the complex mechanisms of DNA interstrand cross-link repair The histone-fold complex MHF is remodeled by FANCM to recognize branched DNA and protect genome stability.Hypersensitivity of primordial germ cells to compromised replication-associated DNA repair involves ATM-p53-p21 signaling RNF4-mediated polyubiquitination regulates the Fanconi anemia/BRCA pathway.DNA damage response factors from diverse pathways, including DNA crosslink repair, mediate alternative end joining.Selective modulation of the functions of a conserved DNA motor by a histone fold complex.FANCM interacts with PCNA to promote replication traverse of DNA interstrand crosslinks.The BLM dissolvasome in DNA replication and repair.A novel interplay between the Fanconi anemia core complex and ATR-ATRIP kinase during DNA cross-link repair.DNA crosslinking damage and cancer - a tale of friend and foe.The DNA translocase FANCM/MHF promotes replication traverse of DNA interstrand crosslinks.DNA repair pathways in human multiple myeloma: role in oncogenesis and potential targets for treatment.Histone deacetylase inhibitors selectively target homology dependent DNA repair defective cells and elevate non-homologous endjoining activity.Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks The differences between ICL repair during and outside of S phase Replication fork regression and its regulation.Fatal Lymphoproliferative Disease in Two Siblings Lacking Functional FAAP24.Biochemical Activities and Genetic Functions of the Drosophila melanogaster Fancm Helicase in DNA Repair.Modularized functions of the Fanconi anemia core complex.FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.Fanconi Anemia Signaling and Cancer.

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FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

http://www.wikidata.org/entity/Q36677983

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@ast

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@en

type

label

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@ast

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@en

prefLabel

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@ast

FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@en

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J.MOLCEL.2012.12.010

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FANCM and FAAP24 maintain genome stability via cooperative as well as unique functions

@en

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Huong Do

http://www.w3.org/2001/XMLSchema#string

Junjie Chen

http://www.w3.org/2001/XMLSchema#string

Justin W Leung

http://www.w3.org/2001/XMLSchema#string

Karen M Vasquez

http://www.w3.org/2001/XMLSchema#string

Lei Li

http://www.w3.org/2001/XMLSchema#string

Megan G Lowery

http://www.w3.org/2001/XMLSchema#string

Weidong Wang

http://www.w3.org/2001/XMLSchema#string

Yingjun Jiang

http://www.w3.org/2001/XMLSchema#string

Yucai Wang

http://www.w3.org/2001/XMLSchema#string

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FAAP100 is essential for activation of the Fanconi anemia-associated DNA damage response pathway

Regulation of Rev1 by the Fanconi anemia core complex

A histone-fold complex and FANCM form a conserved DNA-remodeling complex to maintain genome stability

MHF1-MHF2, a histone-fold-containing protein complex, participates in the Fanconi anemia pathway via FANCM

FAAP20: a novel ubiquitin-binding FA nuclear core-complex protein required for functional integrity of the FA-BRCA DNA repair pathway

Fanconi anemia (FA) binding protein FAAP20 stabilizes FA complementation group A (FANCA) and participates in interstrand cross-link repair

Identification of KIAA1018/FAN1, a DNA repair nuclease recruited to DNA damage by monoubiquitinated FANCD2

Deficiency of FANCD2-associated nuclease KIAA1018/FAN1 sensitizes cells to interstrand crosslinking agents

A genetic screen identifies FAN1, a Fanconi anemia-associated nuclease necessary for DNA interstrand crosslink repair

A ubiquitin-binding protein, FAAP20, links RNF8-mediated ubiquitination to the Fanconi anemia DNA repair network

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997-1009

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scholarly article

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10.1016/J.MOLCEL.2012.12.010

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5

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49

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2013-01-17T00:00:00Z

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234699623

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23333308

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3595374

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