Netrin-1-treated macrophages protect the kidney against ischemia-reperfusion injury and suppress inflammation by inducing M2 polarization.
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Macrophage Phenotype in Kidney Injury and RepairPPARγ and the Innate Immune System Mediate the Resolution of InflammationGuidance cue netrin-1 and the regulation of inflammation in acute and chronic kidney diseaseNetrin-1 ameliorates myocardial infarction-induced myocardial injury: mechanisms of action in rats and diabetic mice.Adoptive transfer of cytokine-induced immunomodulatory adult microglia attenuates experimental autoimmune encephalomyelitis in DBA/1 mice.Malibatol A regulates microglia M1/M2 polarization in experimental stroke in a PPARγ-dependent manner.Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms.Macrophage polarization in kidney diseases.Epoxyeicosatrienoic acids regulate macrophage polarization and prevent LPS-induced cardiac dysfunction.Modulation of Macrophage Polarization and HMGB1-TLR2/TLR4 Cascade Plays a Crucial Role for Cardiac Remodeling in Senescence-Accelerated Prone Mice.Netrin-1 attenuates the progression of renal dysfunction by blocking endothelial-to-mesenchymal transition in the 5/6 nephrectomy rat model.The level of netrin-1 is decreased in newly diagnosed type 2 diabetes mellitus patientsHuman endometrial regenerative cells attenuate renal ischemia reperfusion injury in mice.Blocking Notch signal in myeloid cells alleviates hepatic ischemia reperfusion injury by repressing the activation of NF-κB through CYLDMacrophage-mediated injury and repair after ischemic kidney injury.Immune cells in experimental acute kidney injury.Macrophages in kidney injury, inflammation, and fibrosis.Resolution of inflammation pathways in preeclampsia-a narrative review.Netrin-1 is highly expressed and required in inflammatory infiltrates in wear particle-induced osteolysis.Netrin-1 and its receptor Unc5b are novel targets for the treatment of inflammatory arthritis.Netrin-1 Promotes Inflammation Resolution to Achieve Endothelialization of Small-Diameter Tissue Engineering Blood Vessels by Improving Endothelial Progenitor Cells Function In Situ.Lentiviral-Mediated Netrin-1 Overexpression Improves Motor and Sensory Functions in SCT Rats Associated with SYP and GAP-43 Expressions.Recombinant Netrin-1 binding UNC5B receptor attenuates neuroinflammation and brain injury via PPARγ/NFκB signaling pathway after subarachnoid hemorrhage in rats.Cell-intrinsic sphingosine kinase 2 promotes macrophage polarization and renal inflammation in response to unilateral ureteral obstruction.PPAR-gamma activation is associated with reduced liver ischemia-reperfusion injury and altered tissue-resident macrophages polarization in a mouse model.Netrin-1 promotes diabetic corneal wound healing through molecular mechanisms mediated via the adenosine 2B receptor.Netrin-1 attenuates brain injury after middle cerebral artery occlusion via downregulation of astrocyte activation in mice
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P2860
Netrin-1-treated macrophages protect the kidney against ischemia-reperfusion injury and suppress inflammation by inducing M2 polarization.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@ast
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@en
type
label
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@ast
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@en
prefLabel
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@ast
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@en
P2093
P2860
P1476
Netrin-1-treated macrophages p ...... n by inducing M2 polarization.
@en
P2093
Calpurnia Jayakumar
Ganesan Ramesh
Punithavathi Vilapakkam Ranganathan
P2860
P304
P356
10.1152/AJPRENAL.00580.2012
P577
2013-02-13T00:00:00Z