Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
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Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunction.Cysteine Oxidation Targets Peroxiredoxins 1 and 2 for Exosomal Release through a Novel Mechanism of Redox-Dependent SecretionRedox regulation of mitochondrial functionHigh-Protein Intake during Weight Loss Therapy Eliminates the Weight-Loss-Induced Improvement in Insulin Action in Obese Postmenopausal WomenIdentification of cardiac myosin-binding protein C as a candidate biomarker of myocardial infarction by proteomics analysisCaenorhabditis elegans: A useful model for studying metabolic disorders in which oxidative stress is a contributing factor.Reduction of cysteine sulfinic acid in eukaryotic, typical 2-Cys peroxiredoxins by sulfiredoxin.Nox4 regulates Nrf2 and glutathione redox in cardiomyocytes in vivo.Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunitiesHydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system.Reactive oxygen species and the cardiovascular systemKinetic analysis of structural influences on the susceptibility of peroxiredoxins 2 and 3 to hyperoxidationRoles for cytosolic NADPH redox in regulating pulmonary artery relaxation by thiol oxidation-elicited subunit dimerization of protein kinase G1αMitochondrial peroxiredoxin 3 is more resilient to hyperoxidation than cytoplasmic peroxiredoxins.Protection of peroxiredoxin II on oxidative stress-induced cardiomyocyte death and apoptosis.Mitochondrial peroxiredoxin involvement in antioxidant defence and redox signalling.Contemporary techniques for detecting and identifying proteins susceptible to reversible thiol oxidation.Hydrogen peroxide modifies human sperm peroxiredoxins in a dose-dependent manner.Reversible oxidation of mitochondrial peroxiredoxin 3 in mouse heart subjected to ischemia and reperfusion.THE MULTI-FACETED IMPACT OF PEROXIREDOXINS ON AGING AND DISEASE.Alterations in NO/ROS ratio and expression of Trx1 and Prdx2 in isoproterenol-induced cardiac hypertrophy.Short-term use of telmisartan attenuates oxidation and improves Prdx2 expression more than antioxidant β-blockers in the cardiovascular systems of spontaneously hypertensive rats.Peroxiredoxin as a functional endogenous antioxidant enzyme in pronuclei of mouse zygotes.
P2860
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P2860
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@ast
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@en
type
label
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@ast
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@en
prefLabel
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@ast
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@en
P2093
P2860
P1476
Cardiac peroxiredoxins undergo complex modifications during cardiac oxidant stress.
@en
P2093
Ewald Schröder
Jonathan P Brennan
Philip Eaton
P2860
P304
P356
10.1152/AJPHEART.00017.2008
P577
2008-05-23T00:00:00Z