mTORC1 is necessary but mTORC2 and GSK3β are inhibitory for AKT3-induced axon regeneration in the central nervous system.
about
PKBγ/AKT3 loss-of-function causes learning and memory deficits and deregulation of AKT/mTORC2 signaling: Relevance for schizophrenia.Tyrosine-mutated AAV2-mediated shRNA silencing of PTEN promotes axon regeneration of adult optic nerveRescue of Glaucomatous Neurodegeneration by Differentially Modulating Neuronal Endoplasmic Reticulum Stress Molecules.Rapamycin-Resistant mTOR Activity Is Required for Sensory Axon Regeneration Induced by a Conditioning Lesion.Express yourself: how PP2A-B55(Pab1) helps TORC1 talk to TORC2.The TOR Signaling Pathway in Spatial and Temporal Control of Cell Size and GrowthBoosting CNS axon regeneration by harnessing antagonistic effects of GSK3 activity.A novel mouse model of anterior segment dysgenesis (ASD): conditional deletion of Tsc1 disrupts ciliary body and iris developmentThe age factor in axonal repair after spinal cord injury: A focus on neuron-intrinsic mechanisms.Neuroprotection by eIF2α-CHOP inhibition and XBP-1 activation in EAE/optic neuritiss.Effects of soluble CPE on glioma cell migration are associated with mTOR activation and enhanced glucose flux.Activation of the Akt/mTOR signaling pathway: A potential response to long-term neuronal loss in the hippocampus after sepsis.Optic nerve regeneration in the mouse is a complex trait modulated by genetic background.PTEN negatively regulates the cell lineage progression from NG2+ glial progenitor to oligodendrocyte via mTOR-independent signaling.PTEN knockdown with the Y444F mutant AAV2 vector promotes axonal regeneration in the adult optic nerve.Deletion of the Imprinted Gene Grb10 Promotes Hematopoietic Stem Cell Self-Renewal and Regeneration.Upregulation of neuronal astrocyte elevated gene-1 protects nigral dopaminergic neurons in vivo.Neuronal lysosomes.Beneficial Effects of Resveratrol-Mediated Inhibition of the mTOR Pathway in Spinal Cord Injury.Network analysis of microRNAs, transcription factors, and target genes involved in axon regeneration.Insulin signalling promotes dendrite and synapse regeneration and restores circuit function after axonal injury.Coordination of Necessary and Permissive Signals by PTEN Inhibition for CNS Axon Regeneration
P2860
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P2860
mTORC1 is necessary but mTORC2 and GSK3β are inhibitory for AKT3-induced axon regeneration in the central nervous system.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
mTORC1 is necessary but mTORC2 ...... in the central nervous system.
@en
type
label
mTORC1 is necessary but mTORC2 ...... in the central nervous system.
@en
prefLabel
mTORC1 is necessary but mTORC2 ...... in the central nervous system.
@en
P2093
P2860
P50
P356
P1433
P1476
mTORC1 is necessary but mTORC2 ...... in the central nervous system.
@en
P2093
Feisi Liang
Haoliang Huang
P2860
P304
P356
10.7554/ELIFE.14908
P407
P577
2016-03-30T00:00:00Z