Upregulation of CHOP/GADD153 during coronavirus infectious bronchitis virus infection modulates apoptosis by restricting activation of the extracellular signal-regulated kinase pathway.
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Regulation of Stress Responses and Translational Control by CoronavirusPorcine circovirus type 2 capsid protein induces unfolded protein response with subsequent activation of apoptosis.Coronavirus infection, ER stress, apoptosis and innate immunityER stress, autophagy, and RNA viruses.The endoplasmic reticulum stress sensor IRE1α protects cells from apoptosis induced by the coronavirus infectious bronchitis virusEndoplasmic reticulum stress contributes to Helicobacter pylori VacA-induced apoptosis.Constant up-regulation of BiP/GRP78 expression prevents virus-induced apoptosis in BHK-21 cells with Japanese encephalitis virus persistent infection.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionNiclosamide induced cell apoptosis via upregulation of ATF3 and activation of PERK in Hepatocellular carcinoma cellsThe Important Role of Lipid Raft-Mediated Attachment in the Infection of Cultured Cells by Coronavirus Infectious Bronchitis Virus Beaudette Strain.The NF-κB-dependent and -independent transcriptome and chromatin landscapes of human coronavirus 229E-infected cells.Cadmium and cellular signaling cascades: interactions between cell death and survival pathways.The integrated stress response in lung disease.N-Myc expression enhances the oncolytic effects of vesicular stomatitis virus in human neuroblastoma cellsActivating transcription factor 4 underlies the pathogenesis of arsenic trioxide-mediated impairment of macrophage innate immune functions.Human Coronaviruses: A Review of Virus-Host Interactions.Activation of the c-Jun NH2-terminal kinase pathway by coronavirus infectious bronchitis virus promotes apoptosis independently of c-Jun.Understanding of the functional role(s) of the Activating Transcription Factor 4(ATF4) in HIV regulation and production.The PERK Arm of the Unfolded Protein Response Negatively Regulates Transmissible Gastroenteritis Virus Replication by Suppressing Protein Translation and Promoting Type I IFN Production.The PGRS Domain of Mycobacterium tuberculosis PE_PGRS Protein Rv0297 Is Involved in Endoplasmic Reticulum Stress-Mediated Apoptosis through Toll-Like Receptor 4.
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P2860
Upregulation of CHOP/GADD153 during coronavirus infectious bronchitis virus infection modulates apoptosis by restricting activation of the extracellular signal-regulated kinase pathway.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Upregulation of CHOP/GADD153 d ...... gnal-regulated kinase pathway.
@en
type
label
Upregulation of CHOP/GADD153 d ...... gnal-regulated kinase pathway.
@en
prefLabel
Upregulation of CHOP/GADD153 d ...... gnal-regulated kinase pathway.
@en
P2093
P2860
P356
P1433
P1476
Upregulation of CHOP/GADD153 d ...... gnal-regulated kinase pathway.
@en
P2093
Ding Xiang Liu
Shou Guo Fang
To Sing Fung
Yanxin Zhong
P2860
P304
P356
10.1128/JVI.00626-13
P407
P577
2013-05-15T00:00:00Z