Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
about
A tricyclic antidepressant, amoxapine, reduces amyloid-β generation through multiple serotonin receptor 6-mediated targets.Serotonin augmentation therapy by escitalopram has minimal effects on amyloid-β levels in early-stage Alzheimer's-like disease in mice.Citalopram Ameliorates Impairments in Spatial Memory and Synaptic Plasticity in Female 3xTgAD Mice5-HT6 Receptor Agonist and Antagonist Against β-Amyloid-Peptide-Induced Neurotoxicity in PC-12 Cells.Combination anti-Aβ treatment maximizes cognitive recovery and rebalances mTOR signaling in APP mice.AMPA-ergic regulation of amyloid-β levels in an Alzheimer's disease mouse model.Interactions between stress and physical activity on Alzheimer's disease pathology.Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine.
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Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@en
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@nl
type
label
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@en
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@nl
prefLabel
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@en
Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
@nl
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Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo.
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Clare E Wallace
Danielle L Tripoli
John R Cirrito
Jonathan R Fisher
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P2888
P356
10.1186/S13024-016-0112-5
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2016-06-18T00:00:00Z
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1003954558