Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS. - Wikidata - thesis-toulmin - TriplyDB

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

http://www.wikidata.org/entity/Q37142664

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All muscarinic acetylcholine receptors (M1-M5) are expressed in murine brain microvascular endothelium.Leaky ryanodine receptors contribute to diaphragmatic weakness during mechanical ventilation.Mitochondria and Angiogenesis.IP3 receptor signaling and endothelial barrier function.Vasoconstrictor stimulus determines the functional contribution of myoendothelial feedback to mesenteric arterial tone.Intracellular calcium release channels: an update.Physiology and pathophysiology of excitation-contraction coupling: the functional role of ryanodine receptor.miRNA-145 is associated with spontaneous hypertension by targeting SLC7A1.Ryanodine Receptor Structure and Function in Health and Disease.The Amino-Terminal Domain of GRK5 Inhibits Cardiac Hypertrophy through the Regulation of Calcium-Calmodulin Dependent Transcription Factors.The genetic component of preeclampsia: A whole-exome sequencing study.The Non-Coding RNA Journal Club: Highlights on Recent Papers-4.

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Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

http://www.wikidata.org/entity/Q37142664

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 08 July 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@en

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@nl

type

label

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@en

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@nl

prefLabel

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@en

Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS.

@nl

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PNAS.1608859113

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Proceedings of the National Academy of Sciences of the United States of America

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Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS

@en

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Andrew R Marks

http://www.w3.org/2001/XMLSchema#string

Anetta Wronska

http://www.w3.org/2001/XMLSchema#string

Brent W Osborne

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Jingyi Yang

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Mindy M Kim

http://www.w3.org/2001/XMLSchema#string

Qi Yuan

http://www.w3.org/2001/XMLSchema#string

Steven R Reiken

http://www.w3.org/2001/XMLSchema#string

Yuxin Yin

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Signals transduced by Ca(2+)/calcineurin and NFATc3/c4 pattern the developing vasculature

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Hypertension caused by a truncated epithelial sodium channel gamma subunit: genetic heterogeneity of Liddle syndrome

Vascular endothelial cells synthesize nitric oxide from L-arginine

AT1A angiotensin receptors in the renal proximal tubule regulate blood pressure.

Calcium release channel RyR2 regulates insulin release and glucose homeostasis

Hydrogen peroxide is an endothelium-derived hyperpolarizing factor in mice.

Identification of a family of calcium sensors as protein ligands of inositol trisphosphate receptor Ca(2+) release channels.

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8532-8537

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scholarly article

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10.1073/PNAS.1608859113

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30

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113

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Alain Lacampagne

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2016-07-08T00:00:00Z

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27402766

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4968706

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