Endogenous interleukin-1β in neuropathic rats enhances glutamate release from the primary afferents in the spinal dorsal horn through coupling with presynaptic N-methyl-D-aspartic acid receptors.
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Macrophage repolarization with targeted alginate nanoparticles containing IL-10 plasmid DNA for the treatment of experimental arthritisGlial contributions to visceral pain: implications for disease etiology and the female predominance of persistent painInterleukin-1 beta enhances endocytosis of glial glutamate transporters in the spinal dorsal horn through activating protein kinase C.Spinal inhibition of p38 MAP kinase reduces inflammatory and neuropathic pain in male but not female mice: Sex-dependent microglial signaling in the spinal cord.Pathological pain and the neuroimmune interface.The development and maintenance of paclitaxel-induced neuropathic pain require activation of the sphingosine 1-phosphate receptor subtype 1.Nerve injury-induced epigenetic silencing of opioid receptors controlled by DNMT3a in primary afferent neurons.Activation of toll like receptor 4 attenuates GABA synthesis and postsynaptic GABA receptor activities in the spinal dorsal horn via releasing interleukin-1 beta.Paclitaxel induces acute pain via directly activating toll like receptor 4Adenosine Monophosphate-activated Protein Kinase Regulates Interleukin-1β Expression and Glial Glutamate Transporter Function in Rodents with Neuropathic Pain.Gabapentin prevents oxaliplatin-induced central sensitization in the dorsal horn neurons in ratsActivity-triggered tetrapartite neuron-glial interactions following peripheral injuryHydroxysafflor Yellow A Protects Neurons From Excitotoxic Death through Inhibition of NMDARs.AMPKα1 knockout enhances nociceptive behaviors and spinal glutamatergic synaptic activities via production of reactive oxygen species in the spinal dorsal hornG9a inhibits CREB-triggered expression of mu opioid receptor in primary sensory neurons following peripheral nerve injury.Role of sphingomyelinases in neurological disorders.Toll-like receptors and their role in persistent pain.Increased Central Nervous System Interleukin-8 in a Majority Postlaminectomy Syndrome Chronic Pain Population.Ropivacaine and Bupivacaine prevent increased pain sensitivity without altering neuroimmune activation following repeated social defeat stress.Potentiation of spinal glutamatergic response in the neuron-glia interactions underlies the intrathecal IL-1β-induced thermal hyperalgesia in rats.Chronic pain and impaired glial glutamate transporter function in lupus-prone mice are ameliorated by blocking macrophage colony-stimulating factor-1 receptors.Dysregulation of sphingolipid metabolism contributes to bortezomib-induced neuropathic pain.Neuroinflammation and Central Sensitization in Chronic and Widespread Pain.EZH2 regulates spinal neuroinflammation in rats with neuropathic pain.Human immunodeficiency virus type 1 (HIV-1)-mediated neuroinflammation dysregulates neurogranin and induces synaptodendritic injury.
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Endogenous interleukin-1β in neuropathic rats enhances glutamate release from the primary afferents in the spinal dorsal horn through coupling with presynaptic N-methyl-D-aspartic acid receptors.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 September 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@en
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@nl
type
label
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@en
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@nl
prefLabel
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@en
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
@nl
P2860
P356
P1476
Endogenous interleukin-1β in n ...... hyl-D-aspartic acid receptors.
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P2093
Han-Rong Weng
Xisheng Yan
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P304
30544-30557
P356
10.1074/JBC.M113.495465
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P577
2013-09-03T00:00:00Z