cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
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Cerebrovascular responses in mice deficient in the potassium channel, TREK-1.TWIK-2 channel deficiency leads to pulmonary hypertension through a rho-kinase-mediated process.Relative contribution of SKCa and TREK1 channels in purinergic and nitrergic neuromuscular transmission in the rat colon.The role of TREK-1 in cerebrovascular regulation remains elusive: focus on "Cerebrovascular responses in mice deficient in the potassium channel, TREK-1".Restoration of the response of the middle cerebral artery of the rat to acidosis in hyposmotic hyponatremia by the opener of large-conductance calcium sensitive potassium channels (BKCa).
P2860
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@en
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@nl
type
label
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@en
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@nl
prefLabel
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@en
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@nl
P2093
P2860
P1476
cGMP does not activate two-pore domain K+ channels in cerebrovascular smooth muscle.
@en
P2093
Eric E Lloyd
Robert M Bryan
Sean P Marrelli
P2860
P304
P356
10.1152/AJPHEART.00082.2009
P577
2009-04-10T00:00:00Z