Haemophilus influenzae from patients with chronic obstructive pulmonary disease exacerbation induce more inflammation than colonizers. - Wikidata - thesis-toulmin - TriplyDB

Haemophilus influenzae from patients with chronic obstructive pulmonary disease exacerbation induce more inflammation than colonizers.

Haemophilus influenzae from patients with chronic obstructive pulmonary disease exacerbation induce more inflammation than colonizers.

http://www.wikidata.org/entity/Q37282727

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Lung microbiology and exacerbations in COPD Advances in understanding COPD A mouse model of lethal synergism between influenza virus and Haemophilus influenzae.Role of the nuclease of nontypeable Haemophilus influenzae in dispersal of organisms from biofilms.Characterization of nontypable Haemophilus influenzae isolates recovered from adult patients with underlying chronic lung disease reveals genotypic and phenotypic traits associated with persistent infection.Prevalence of the sodC gene in nontypeable Haemophilus influenzae and Haemophilus haemolyticus by microarray-based hybridization.Significance of the microbiome in chronic obstructive pulmonary disease.Inhibition of IFN-gamma-dependent antiviral airway epithelial defense by cigarette smoke.Development and characterization of a long-term murine model of Streptococcus pneumoniae infection of the lower airways.Lower airway colonization and inflammatory response in COPD: a focus on Haemophilus influenzae Microbiome diversity in the bronchial tracts of patients with chronic obstructive pulmonary disease.Vitamin D decreases respiratory syncytial virus induction of NF-kappaB-linked chemokines and cytokines in airway epithelium while maintaining the antiviral state Invasive Haemophilus influenzae in Manitoba, Canada, in the postvaccination era Inhibition by cigarette smoke of nuclear factor-κB-dependent response to bacteria in the airway.Novel insights into the aetiology and pathophysiology of increased airway inflammation during COPD exacerbations.Nontypeable Haemophilus Influenzae Infection Upregulates the NLRP3 Inflammasome and Leads to Caspase-1-Dependent Secretion of Interleukin-1β - A Possible Pathway of Exacerbations in COPD The existence of bronchiectasis predicts worse prognosis in patients with COPD.Haemophilus influenzae induces steroid-resistant inflammatory responses in COPD.Experimental therapeutics of Nrf2 as a target for prevention of bacterial exacerbations in COPD Characterization of extended co-culture of non-typeable Haemophilus influenzae with primary human respiratory tissues.Dps promotes survival of nontypeable Haemophilus influenzae in biofilm communities in vitro and resistance to clearance in vivo Role of infection and antimicrobial therapy in acute exacerbations of chronic obstructive pulmonary disease.COPD exacerbations . 2: aetiology Emerging drugs for the treatment of chronic obstructive pulmonary disease.Update in chronic obstructive pulmonary disease 2005.Pathogenesis of bacterial exacerbations of COPD.Pathogen-directed therapy in acute exacerbations of chronic obstructive pulmonary disease.Optimizing antibiotic selection in treating COPD exacerbations Why we need a vaccine for non-typeable Haemophilus influenzae.Role of macrolide therapy in chronic obstructive pulmonary disease.Recognizing the importance of respiratory syncytial virus in chronic obstructive pulmonary disease.Differential effects of cytokines and corticosteroids on toll-like receptor 2 expression and activity in human airway epithelia.Update in chronic obstructive pulmonary disease 2009.Phenotyping the heterogeneity of chronic obstructive pulmonary disease.Nontypeable Haemophilus influenzae and chronic obstructive pulmonary disease: a review for clinicians.Non-typeable Haemophilus influenzae protects human airway epithelial cells from a subsequent respiratory syncytial virus challenge.Diversity of nontypeable Haemophilus influenzae strains colonizing Australian Aboriginal and non-Aboriginal children.Secondary Bacterial Infections Associated with Influenza Pandemics The T-helper cell type 1 immune response to gram-negative bacterial infections is impaired in COPD.Moraxella catarrhalis induces inflammatory response of bronchial epithelial cells via MAPK and NF-kappaB activation and histone deacetylase activity reduction.

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Haemophilus influenzae from patients with chronic obstructive pulmonary disease exacerbation induce more inflammation than colonizers.

http://www.wikidata.org/entity/Q37282727

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bilimsel makale

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scientific article published on April 2005

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vetenskaplig artikel

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vědecký článek

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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RCCM.200412-1687OC

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American Journal of Respiratory and Critical Care Medicine

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Haemophilus influenzae from pa ...... inflammation than colonizers.

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Alicia L Humlicek

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Cecilia L Chin

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Dwight C Look

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Erin E Lehman

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Gerene M Denning

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Lei Shi

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Lori J Manzel

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Sanjay Sethi

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Timothy D Starner

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P2860

Direct suppression of Stat1 function during adenoviral infection

Biofilm formation by nontypeable Haemophilus influenzae: strain variability, outer membrane antigen expression and role of pili.

Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation

Initiation of biofilm formation in Pseudomonas fluorescens WCS365 proceeds via multiple, convergent signalling pathways: a genetic analysis

Nontypeable Haemophilus influenzae strain 2019 produces a biofilm containing N-acetylneuraminic acid that may mimic sialylated O-linked glycans

Molecular determinants of the pathogenesis of disease due to non-typable Haemophilus influenzae.

Do bacteria cause exacerbations of COPD?

The role of bacteria in exacerbations of COPD. A constructive view.

Adhesin expression in matched nasopharyngeal and middle ear isolates of nontypeable Haemophilus influenzae from children with acute otitis media.

The pathogenesis of nontypable Haemophilus influenzae otitis media.

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10.1164/RCCM.200412-1687OC

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Timothy F. Murphy

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chronic obstructive pulmonary disease

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