Insulin-like growth factor-2 (IGF2) loss of imprinting marks a field defect within human prostates containing cancer.
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Epithelial cancers in the post-genomic era: should we reconsider our lifestyle?Does milk intake promote prostate cancer initiation or progression via effects on insulin-like growth factors (IGFs)? A systematic review and meta-analysisInsulin-like growth factor pathway genetic polymorphisms, circulating IGF1 and IGFBP3, and prostate cancer survival.Epigenetic susceptibility factors for prostate cancer with aging.Dysregulated methylation at imprinted genes in prostate tumor tissue detected by methylation microarray.Single nucleotide polymorphism array profiling of adrenocortical tumors--evidence for an adenoma carcinoma sequence?Role of diet in prostate cancer: the epigenetic link.Association of cord blood methylation fractions at imprinted insulin-like growth factor 2 (IGF2), plasma IGF2, and birth weightRecurrent variations in DNA methylation in human pluripotent stem cells and their differentiated derivatives.Methylation profiling defines an extensive field defect in histologically normal prostate tissues associated with prostate cancerLoss of imprinting of IGF2 and the epigenetic progenitor model of cancerDNA methylation as clinically useful biomarkers-light at the end of the tunnelInsulin Receptor Isoform Variations in Prostate Cancer Cells.Maternal high-fat diet induces hyperproliferation and alters Pten/Akt signaling in prostates of offspring.Specific changes in the expression of imprinted genes in prostate cancer--implications for cancer progression and epigenetic regulation.Prostate cancer epigenetic biomarkers: next-generation technologies.The epigenetics of prostate cancer diagnosis and prognosis: update on clinical applications.Widespread telomere instability in prostatic lesions.Deregulation of an imprinted gene network in prostate cancer.Construction of a lncRNA-PCG bipartite network and identification of cancer-related lncRNAs: a case study in prostate cancer.Even-skipped homeobox 1 is frequently hypermethylated in prostate cancer and predicts PSA recurrenceID4 is frequently downregulated and partially hypermethylated in prostate cancer.Impairment of IGF2 gene expression in prostate cancer is triggered by epigenetic dysregulation of IGF2-DMR0 and its interaction with KLF4.Using the epigenetic field defect to detect prostate cancer in biopsy negative patients.CpG island hypermethylation frequently silences FILIP1L isoform 2 expression in prostate cancer.H19 DMR methylation correlates to the progression of esophageal squamous cell carcinoma through IGF2 imprinting pathway.Insulin-like growth factor 2 expression in prostate cancer is regulated by promoter-specific methylation.Insulin-like Growth Factor Pathway Genetic Polymorphisms, Circulating IGF1 and IGFBP3, and Prostate Cancer Survival.A comprehensive overview of genomic imprinting in breast and its deregulation in cancer
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P2860
Insulin-like growth factor-2 (IGF2) loss of imprinting marks a field defect within human prostates containing cancer.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 22 March 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Insulin-like growth factor-2 ( ...... n prostates containing cancer.
@en
Insulin-like growth factor-2
@nl
type
label
Insulin-like growth factor-2 ( ...... n prostates containing cancer.
@en
Insulin-like growth factor-2
@nl
prefLabel
Insulin-like growth factor-2 ( ...... n prostates containing cancer.
@en
Insulin-like growth factor-2
@nl
P2093
P2860
P356
P1433
P1476
Insulin-like growth factor-2 ( ...... n prostates containing cancer.
@en
P2093
David F Jarrard
Jessica Kueck
Sachin Bhusari
P2860
P304
P356
10.1002/PROS.21379
P577
2011-03-22T00:00:00Z