Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
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Recombinant expression of the voltage-dependent anion channel enhances the transfer of Ca2+ microdomains to mitochondriaMitochondrial fusion is frequent in skeletal muscle and supports excitation-contraction couplingPrimary Murine Myotubes as a Model for Investigating Muscular DystrophyTargeted recombinant aequorins: tools for monitoring [Ca2+] in the various compartments of a living cell.Seeing is believing: recent trends in the measurement of Ca2+ in subcellular domains and intracellular organelles.The renaissance of mitochondrial calcium transport.The machinery of local Ca2+ signalling between sarco-endoplasmic reticulum and mitochondria.Recombinant aequorin and green fluorescent protein as valuable tools in the study of cell signallingVoltage clamp methods for the study of membrane currents and SR Ca(2+) release in adult skeletal muscle fibresThe mitochondrial calcium uniporter controls skeletal muscle trophism in vivo.Role of mitochondrial Ca2+ in the regulation of cellular energeticsIn vivo monitoring of Ca(2+) uptake into mitochondria of mouse skeletal muscle during contractionHigh glucose induces adipogenic differentiation of muscle-derived stem cells.Energy sensing and regulation of gene expression in skeletal muscle.Regulation of mitochondrial ATP synthesis by calcium: evidence for a long-term metabolic priming.Interactions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle - pivotal roles in Ca²⁺ and reactive oxygen species signaling.Compartmentalized signalling: Ca2+ compartments, microdomains and the many facets of Ca2+ signalling.Altered Ca2+ homeostasis and endoplasmic reticulum stress in myotonic dystrophy type 1 muscle cells.Mitochondrial Ca(2+)-induced Ca(2+) release mediated by the Ca(2+) uniporter.Physical exercise in aging human skeletal muscle increases mitochondrial calcium uniporter expression levels and affects mitochondria dynamics.Switch from ER-mitochondrial to SR-mitochondrial calcium coupling during muscle differentiation.Transfer and tunneling of Ca2+ from sarcoplasmic reticulum to mitochondria in skeletal muscle.Respiratory complex I dysfunction due to mitochondrial DNA mutations shifts the voltage threshold for opening of the permeability transition pore toward resting levels.Ca2+ signaling in HEK-293 and skeletal muscle cells expressing recombinant ryanodine receptors harboring malignant hyperthermia and central core disease mutations.Calcium-regulated changes in mitochondrial phenotype in skeletal muscle cells.A direct mass-action mechanism explains capacitative calcium entry in Jurkat and skeletal L6 muscle cells.The creatine kinase system is essential for optimal refill of the sarcoplasmic reticulum Ca2+ store in skeletal muscle.New light on mitochondrial calcium.The Golgi apparatus is an inositol 1,4,5-trisphosphate-sensitive Ca2+ store, with functional properties distinct from those of the endoplasmic reticulum.Effect of inositol 1,4,5-trisphosphate receptor stimulation on mitochondrial [Ca2+] and secretion in chromaffin cells.Reactive oxygen species activate a Ca2+-dependent cell death pathway in the unicellular organism Trypanosoma brucei brucei.Nitric oxide inhibition of Drp1-mediated mitochondrial fission is critical for myogenic differentiation.Calcium-insensitive splice variants of mammalian E1 subunit of 2-oxoglutarate dehydrogenase complex with tissue-specific patterns of expression.Control of secretion by mitochondria depends on the size of the local [Ca2+] after chromaffin cell stimulation.Post-natal developmental expression of alphaKAP splice variants in rabbit fast-twitch and slow-twitch skeletal muscle.Subcellular calcium measurements in mammalian cells using jellyfish photoprotein aequorin-based probes.Studies on the regulation of the human E1 subunit of the 2-oxoglutarate dehydrogenase complex, including the identification of a novel calcium-binding site.Protein Kinase C β: a New Target Therapy to Prevent the Long-Term Atypical Antipsychotic-Induced Weight Gain.S-adenosylmethionine stimulates fatty acid metabolism-linked gene expression in porcine muscle satellite cells.Mitochondrial calcium signalling in cell death.
P2860
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P2860
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@en
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@nl
type
label
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@en
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@nl
prefLabel
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@en
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@nl
P2093
P2860
P356
P1476
Subcellular analysis of Ca2+ homeostasis in primary cultures of skeletal muscle myotubes.
@en
P2093
F De Giorgi
M L Massimino
R Marsault
P2860
P304
P356
10.1091/MBC.8.1.129
P577
1997-01-01T00:00:00Z