X11beta rescues memory and long-term potentiation deficits in Alzheimer's disease APPswe Tg2576 mice.

X11beta rescues memory and long-term potentiation deficits in Alzheimer's disease APPswe Tg2576 mice.

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http://www.wikidata.org/entity/Q37411775
Loss of c-Jun N-terminal kinase-interacting protein-1 does not affect axonal transport of the amyloid precursor protein or Aβ production.Calsyntenin-1 mediates axonal transport of the amyloid precursor protein and regulates Aβ productionPhosphorylation of kinesin light chain 1 at serine 460 modulates binding and trafficking of calsyntenin-1.Longitudinal testing of hippocampal plasticity reveals the onset and maintenance of endogenous human Aß-induced synaptic dysfunction in individual freely behaving pre-plaque transgenic rats: rapid reversal by anti-Aß agents.Cytoplasmic fragment of Alcadein α generated by regulated intramembrane proteolysis enhances amyloid β-protein precursor (APP) transport into the late secretory pathway and facilitates APP cleavage.Modifications of hippocampal circuits and early disruption of adult neurogenesis in the tg2576 mouse model of Alzheimer's disease.Systematic review of the relationship between amyloid-β levels and measures of transgenic mouse cognitive deficit in Alzheimer's disease.Hyperpolarization-activated cyclic nucleotide gated channels: a potential molecular link between epileptic seizures and Aβ generation in Alzheimer's diseaseX11/Mint genes control polarized localization of axonal membrane proteins in vivoEarly and sustained altered expression of aging-related genes in young 3xTg-AD mice.Hippocampal synaptic plasticity in Alzheimer's disease: what have we learned so far from transgenic models?Age-related synaptic dysfunction in Tg2576 mice starts as a failure in early long-term potentiation which develops into a full abolishment of late long-term potentiation.Increased amyloidogenic processing of transgenic human APP in X11-like deficient mouse brain.Expression of the neuronal adaptor protein X11alpha protects against memory dysfunction in a transgenic mouse model of Alzheimer's disease.Models of dementia: an introductory overview.Association of Neuropathological Markers in the Parietal Cortex With Antemortem Cognitive Function in Persons With Mild Cognitive Impairment and Alzheimer Disease.Detection of Genomic Imbalances by Array-Based Comparative Genomic Hybridization in Bulgarian Patients with Autism Spectrum Disorders
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X11beta rescues memory and long-term potentiation deficits in Alzheimer's disease APPswe Tg2576 mice.

Item
http://www.wikidata.org/entity/Q37411775
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 10 September 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@en
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@nl
type
label
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@en
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@nl
prefLabel
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@en
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P921
P932
P356
P1433
P1476
X11beta rescues memory and lon ...... 's disease APPswe Tg2576 mice.
@en
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P921
P932