Expression of TMEM106B, the frontotemporal lobar degeneration-associated protein, in normal and diseased human brain.
about
The FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomesCognitive reserve and TMEM106B genotype modulate brain damage in presymptomatic frontotemporal dementia: a GENFI study.TMEM106B expression is reduced in Alzheimer's disease brainsTMEM106B, a frontotemporal lobar dementia (FTLD) modifier, associates with FTD-3-linked CHMP2B, a complex of ESCRT-III.Increased expression of the frontotemporal dementia risk factor TMEM106B causes C9orf72-dependent alterations in lysosomes.What we know about TMEM106B in neurodegeneration.TMEM106B drives lung cancer metastasis by inducing TFEB-dependent lysosome synthesis and secretion of cathepsins.
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Expression of TMEM106B, the frontotemporal lobar degeneration-associated protein, in normal and diseased human brain.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 11 July 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Expression of TMEM106B, the fr ...... rmal and diseased human brain.
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Alice S Chen-Plotkin
Emily Ashbridge
Fenghua Hu
Johanna I Busch
Maria Martinez-Lage
Virginia M Y Lee
Vivianna M Van Deerlin
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10.1186/2051-5960-1-36
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2013-07-11T00:00:00Z
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1006086066