Autophagy, TGF-β, and SMAD-2/3 Signaling Regulates Interferon-β Response in Respiratory Syncytial Virus Infected Macrophages.
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Lethal concentration of perfluoroisobutylene induces acute lung injury in mice mediated via cytokine storm, oxidative stress and apoptosis.Apoptosis, Toll-like, RIG-I-like and NOD-like Receptors Are Pathways Jointly Induced by Diverse Respiratory Bacterial and Viral Pathogens.RSV replication is promoted by autophagy-mediated inhibition of apoptosis.Phosphoproteomic analysis reveals Smad protein family activation following Rift Valley fever virus infection.Autophagy enhances the replication of Peste des petits ruminants virus and inhibits caspase-dependent apoptosis in vitro
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Autophagy, TGF-β, and SMAD-2/3 Signaling Regulates Interferon-β Response in Respiratory Syncytial Virus Infected Macrophages.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 December 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
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Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
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type
label
Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
@en
Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
@nl
prefLabel
Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
@en
Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
@nl
P2093
P2860
P356
P1476
Autophagy, TGF-β, and SMAD-2/3 ...... al Virus Infected Macrophages.
@en
P2093
Niraj K Shil
Santanu Bose
Swechha M Pokharel
P2860
P356
10.3389/FCIMB.2016.00174
P577
2016-12-12T00:00:00Z