During lytic infection herpes simplex virus type 1 is associated with histones bearing modifications that correlate with active transcription.
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Autorepression of the human cytomegalovirus major immediate-early promoter/enhancer at late times of infection is mediated by the recruitment of chromatin remodeling enzymes by IE86CTCF-dependent chromatin boundary element between the latency-associated transcript and ICP0 promoters in the herpes simplex virus type 1 genomeRole of ND10 nuclear bodies in the chromatin repression of HSV-1Potential function of miRNAs in herpetic stromal keratitisHerpes simplex virus type 1 latency and reactivation: an updateThe molecular basis of herpes simplex virus latencyIFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modificationsAn Epigenetic Compound Library Screen Identifies BET Inhibitors That Promote HSV-1 and -2 Replication by Bridging P-TEFb to Viral Gene Promoters through BRD4Genital Herpes: Insights into Sexually Transmitted Infectious DiseaseNeuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.The varicella-zoster virus immediate-early 63 protein affects chromatin-controlled gene transcription in a cell-type dependent mannerRole for A-type lamins in herpesviral DNA targeting and heterochromatin modulationRole of chromatin during herpesvirus infections.Transcriptional coactivator HCF-1 couples the histone chaperone Asf1b to HSV-1 DNA replication componentsHerpes simplex virus VP16, but not ICP0, is required to reduce histone occupancy and enhance histone acetylation on viral genomes in U2OS osteosarcoma cells.Control of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.Chromatin assembly on herpes simplex virus genomes during lytic infection.Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1.Alterations in the Aedes aegypti transcriptome during infection with West Nile, dengue and yellow fever viruses.Dengue virus capsid protein binds core histones and inhibits nucleosome formation in human liver cells.The chromatin remodeling factor SMARCB1 forms a complex with human cytomegalovirus proteins UL114 and UL44Complex modulation of the Aedes aegypti transcriptome in response to dengue virus infection.Cellular SNF2H chromatin-remodeling factor promotes herpes simplex virus 1 immediate-early gene expression and replication.Epigenetic regulation of varicella-zoster virus open reading frames 62 and 63 in latently infected human trigeminal ganglia.Downregulation of varicella-zoster virus (VZV) immediate-early ORF62 transcription by VZV ORF63 correlates with virus replication in vitro and with latency.Quantitative Analysis of HSV Gene Expression during Lytic Infection.Curcumin inhibits herpes simplex virus immediate-early gene expression by a mechanism independent of p300/CBP histone acetyltransferase activity.Tegument protein control of latent herpesvirus establishment and animation.Herpes simplex virus ICP0 promotes both histone removal and acetylation on viral DNA during lytic infection.Upregulation of mouse genes in HSV-1 latent TG after butyrate treatment implicates the multiple roles of the LAT-ICP0 locus.Biphasic euchromatin-to-heterochromatin transition on the KSHV genome following de novo infection.Host-viral effects of chromatin assembly factor 1 interaction with HCMV IE2Histone H3 interacts and colocalizes with the nuclear shuttle protein and the movement protein of a geminivirus.Dynamic association of gammaherpesvirus DNA with core histone during de novo lytic infection of primary cells.Genome wide nucleosome mapping for HSV-1 shows nucleosomes are deposited at preferred positions during lytic infection.Selective recruitment of nuclear factors to productively replicating herpes simplex virus genomes.Barrier-to-Autointegration Factor 1 (BAF/BANF1) Promotes Association of the SETD1A Histone Methyltransferase with Herpes Simplex Virus Immediate-Early Gene Promoters.Roles of the nuclear lamina in stable nuclear association and assembly of a herpesviral transactivator complex on viral immediate-early genes.In vivo changes in the patterns of chromatin structure associated with the latent herpes simplex virus type 1 genome in mouse trigeminal ganglia can be detected at early times after butyrate treatment.Herpesviral ICP0 Protein Promotes Two Waves of Heterochromatin Removal on an Early Viral Promoter during Lytic Infection.
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During lytic infection herpes simplex virus type 1 is associated with histones bearing modifications that correlate with active transcription.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
During lytic infection herpes ...... ate with active transcription.
@en
During lytic infection herpes ...... ate with active transcription.
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type
label
During lytic infection herpes ...... ate with active transcription.
@en
During lytic infection herpes ...... ate with active transcription.
@nl
prefLabel
During lytic infection herpes ...... ate with active transcription.
@en
During lytic infection herpes ...... ate with active transcription.
@nl
P2093
P2860
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During lytic infection herpes ...... late with active transcription
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P2860
P304
10178-10186
P356
10.1128/JVI.78.18.10178-10186.2004
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P577
2004-09-01T00:00:00Z