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Mec1/ATR, the Program Manager of Nucleic Acids Inc.

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

http://www.wikidata.org/entity/Q37628254

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The Role of the Transcriptional Response to DNA Replication Stress.The DNA Pol ϵ stimulatory activity of Mrc1 is modulated by phosphorylation.

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Mec1/ATR, the Program Manager of Nucleic Acids Inc.

http://www.wikidata.org/entity/Q37628254

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 December 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

@en

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

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type

label

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

@en

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

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prefLabel

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

@en

Mec1/ATR, the Program Manager of Nucleic Acids Inc.

@nl

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Mec1/ATR, the Program Manager of Nucleic Acids Inc

@en

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Wenyi Feng

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ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks

Nucleolar responses to DNA double-strand breaks

Replication and transcription on a collision course: eukaryotic regulation mechanisms and implications for DNA stability

Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes

Co-transcriptional degradation by the 5'-3' exonuclease Rat1p mediates quality control of HXK1 mRNP biogenesis in S. cerevisiae

NORF5/HUG1 is a component of the MEC1-mediated checkpoint response to DNA damage and replication arrest in Saccharomyces cerevisiae.

A role for Saccharomyces cerevisiae histone H2A in DNA repair.

Rad53-Mediated Regulation of Rrm3 and Pif1 DNA Helicases Contributes to Prevention of Aberrant Fork Transitions under Replication Stress.

The Dun1 checkpoint kinase phosphorylates and regulates the ribonucleotide reductase inhibitor Sml1.

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scholarly article

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10.3390/GENES8010010

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1

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8

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2016-12-28T00:00:00Z

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5295005

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