Enhanced p22phox expression impairs vascular function through p38 and ERK1/2 MAP kinase-dependent mechanisms in type 2 diabetic mice.
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The Role of p38 MAPK in the Development of Diabetic Cardiomyopathyp38 MAPK in cardioprotection - are we there yet?Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse heartsTissue-specific up-regulation of arginase I and II induced by p38 MAPK mediates endothelial dysfunction in type 1 diabetes mellitus.Endothelial dysfunction and vascular disease - a 30th anniversary update.Microvascular NADPH oxidase in health and disease.ROS Signaling in the Pathogenesis of Acute Lung Injury (ALI) and Acute Respiratory Distress Syndrome (ARDS).Endothelial Cell Autophagy Maintains Shear Stress-Induced Nitric Oxide Generation via Glycolysis-Dependent Purinergic Signaling to Endothelial Nitric Oxide Synthase.
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Enhanced p22phox expression impairs vascular function through p38 and ERK1/2 MAP kinase-dependent mechanisms in type 2 diabetic mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 31 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
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Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
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type
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Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
@en
Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
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Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
@en
Enhanced p22phox expression im ...... nisms in type 2 diabetic mice.
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Enhanced p22phox expression im ...... anisms in type 2 diabetic mice
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Khalid Matrougui
Modar Kassan
Mohamed Trebak
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10.1152/AJPHEART.00872.2013
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2014-01-31T00:00:00Z