Amotl1 mediates sequestration of the Hippo effector Yap1 downstream of Fat4 to restrict heart growth. - Wikidata - thesis-toulmin - TriplyDB

Amotl1 mediates sequestration of the Hippo effector Yap1 downstream of Fat4 to restrict heart growth.

Amotl1 mediates sequestration of the Hippo effector Yap1 downstream of Fat4 to restrict heart growth.

http://www.wikidata.org/entity/Q37676704

about

Angiomotin Family Members: Oncogenes or Tumor Suppressors?Wnt Signalling Drives Context-Dependent Differentiation or Proliferation in Neuroblastoma.Tissue-specific activities of the Fat1 cadherin cooperate to control neuromuscular morphogenesis.

P2860

Q33837277-D6D48CA3-C474-4A53-B8EC-ED26975FFC01 Q52673795-AC618D99-3532-4141-93D7-265F5B02B6A8 Q55094954-2B334DB9-5751-4E9A-A66A-D094E4B4E531

P2860

Amotl1 mediates sequestration of the Hippo effector Yap1 downstream of Fat4 to restrict heart growth.

http://www.wikidata.org/entity/Q37676704

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 27 February 2017

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@en

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@nl

type

label

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@en

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@nl

prefLabel

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@en

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@nl

P1433

Q37676704-20ECF9B0-5F3F-4F6F-BBAF-568C2850EF6E

P1476

Q37676704-AA18A638-323C-4B7B-9675-45D1DBDAD093

P2093

Q37676704-4423F714-2831-4110-8775-C51ACCB08877

Q37676704-5C4D87B9-B03A-499B-99FD-45A2D01F424B

Q37676704-67D352CD-32BD-4E0E-BDF2-6DBD182D4EC9

Q37676704-A0C5E256-0A0C-4F7E-A51C-D262C8E20BBA

Q37676704-BC91D305-3202-49D3-8C31-32A06F17059E

Q37676704-E28F0454-3B15-48E2-8062-89DE44792098

P2860

Q37676704-02204389-6C9B-4C66-972D-8C6021ECCE47

Q37676704-07EF8623-F690-4D40-B456-1B57B03B2DE9

Q37676704-0CBCA0AA-343D-4106-A3C2-F26A228449D2

Q37676704-121FB2F5-BA1E-47E0-A973-1D424A30A9F2

Q37676704-1669AEE3-88F9-49D5-8307-080BB386E150

Q37676704-203F2945-265C-44FF-A191-9DA65FF48695

Q37676704-2CE395EC-A43B-4E93-9D7A-F02021E79660

Q37676704-397DD77C-1588-4AC6-B3F4-17A6F1075455

Q37676704-3F731326-79B8-4D4D-A131-E52694F34569

Q37676704-401E7E8F-29B0-447E-B207-E923DF70F298

P2888

Q37676704-1F1D5B6F-9A3E-4607-BE9B-11B734DE9D6B

P304

Q37676704-4080C10E-35B7-4AE3-8236-FDFFE5A60F64

P31

Q37676704-EB802E0E-E271-4147-956F-D197D54D5FA5

P356

Q37676704-369E0CA1-2FC0-4773-B1D5-78605236E123

P407

Q37676704-28ADB71A-67F1-41A3-86AA-D2606E533D32

P478

Q37676704-2F3768AF-4BC6-4AF1-8F20-EF778C1C0640

P50

Q37676704-03980BA3-4629-4197-B880-8FA3EC099EAA

Q37676704-0CFABE20-B230-4F88-B50E-A6DCE9DC0281

Q37676704-0DCD5DDD-8094-4E5F-935C-DD7F29340AAA

Q37676704-7E3329F3-8787-484D-9BD1-8AFCBBB6BED6

Q37676704-8DB9BF36-CF33-4425-8658-4B70ACE5D0DC

Q37676704-9FA6FA7E-0432-4F50-B078-538A6E4CB1A1

Q37676704-CE8A524A-AF49-45D5-9A24-EBDE03991CF4

Q37676704-D4DD67DE-D6FD-49C3-AE30-CC3BDA425914

Q37676704-EFEDCF4D-712B-4050-AEDC-4470C964B33B

P577

Q37676704-9C241E83-CF6C-4C7C-B7F2-F2892D906614

P6179

Q37676704-9002D2D6-9A12-4E49-B0F2-3F319D8F83D8

P698

Q37676704-DA014BCD-2F21-44F4-85FF-CEBB83E86C00

P932

Q37676704-8B379578-4E08-487D-9BB3-ECE170A682E4

P356

P1433

Nature Communications

P1476

Amotl1 mediates sequestration ...... Fat4 to restrict heart growth.

@en

P2093

Alain Trouvé

http://www.w3.org/2001/XMLSchema#string

Caroline Badouel

http://www.w3.org/2001/XMLSchema#string

Jean-Christophe Olivo-Marin

http://www.w3.org/2001/XMLSchema#string

Marta Novotova

http://www.w3.org/2001/XMLSchema#string

Nicolas Charon

http://www.w3.org/2001/XMLSchema#string

Sorin Pop

http://www.w3.org/2001/XMLSchema#string

P2860

Angiomotin-like proteins associate with and negatively regulate YAP1

Angiomotin is a novel Hippo pathway component that inhibits YAP oncoprotein

Yap- and Cdc42-dependent nephrogenesis and morphogenesis during mouse kidney development

The Hippo signaling pathway coordinately regulates cell proliferation and apoptosis by inactivating Yorkie, the Drosophila Homolog of YAP

Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload

Hippo pathway inhibits Wnt signaling to restrain cardiomyocyte proliferation and heart size

Characterization of a Dchs1 mutant mouse reveals requirements for Dchs1-Fat4 signaling during mammalian development

Mammalian Fat and Dachsous cadherins regulate apical membrane organization in the embryonic cerebral cortex

Molecular characterization of angiomotin/JEAP family proteins: interaction with MUPP1/Patj and their endogenous properties

MesP1 is expressed in the heart precursor cells and required for the formation of a single heart tube

P2888

P304

14582

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/NCOMMS14582

http://www.w3.org/2001/XMLSchema#string

P407

P478

8

http://www.w3.org/2001/XMLSchema#string

P50

Tatiana P. Resende

Laurent Guillemot

Sigolène M Meilhac

Alexandre Dufour

Jean-Francois Le Garrec

P577

2017-02-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1084128827

http://www.w3.org/2001/XMLSchema#string

P698

28239148

http://www.w3.org/2001/XMLSchema#string

P932

5333361

http://www.w3.org/2001/XMLSchema#string