Increased PLEKHO1 within osteoblasts suppresses Smad-dependent BMP signaling to inhibit bone formation during aging. - Wikidata - thesis-toulmin - TriplyDB

Increased PLEKHO1 within osteoblasts suppresses Smad-dependent BMP signaling to inhibit bone formation during aging.

Increased PLEKHO1 within osteoblasts suppresses Smad-dependent BMP signaling to inhibit bone formation during aging.

http://www.wikidata.org/entity/Q37678213

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The role of CKIP-1 in osteoporosis development and treatment.

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Increased PLEKHO1 within osteoblasts suppresses Smad-dependent BMP signaling to inhibit bone formation during aging.

http://www.wikidata.org/entity/Q37678213

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 13 January 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Increased PLEKHO1 within osteo ...... t bone formation during aging.

@en

Increased PLEKHO1 within osteo ...... t bone formation during aging.

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type

label

Increased PLEKHO1 within osteo ...... t bone formation during aging.

@en

Increased PLEKHO1 within osteo ...... t bone formation during aging.

@nl

prefLabel

Increased PLEKHO1 within osteo ...... t bone formation during aging.

@en

Increased PLEKHO1 within osteo ...... t bone formation during aging.

@nl

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Increased PLEKHO1 within osteo ...... it bone formation during aging

@en

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Aiping Lu

http://www.w3.org/2001/XMLSchema#string

Bao-Ting Zhang

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Changwei Lu

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Chao Liang

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Defang Li

http://www.w3.org/2001/XMLSchema#string

Ge Zhang

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Jin Liu

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Kang Zheng

http://www.w3.org/2001/XMLSchema#string

Lei Dang

http://www.w3.org/2001/XMLSchema#string

Songlin Peng

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P2860

Regulation of Smad degradation and activity by Smurf2, an E3 ubiquitin ligase

Ubiquitin C-terminal hydrolase-L3 regulates Smad1 ubiquitination and osteoblast differentiation

TGF-β and BMP signaling in osteoblast differentiation and bone formation

Targeting WW domains linker of HECT-type ubiquitin ligase Smurf1 for activation by CKIP-1

Sclerostin antibody treatment increases bone formation, bone mass, and bone strength in a rat model of postmenopausal osteoporosis

BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation

Ubiquitin ligase Smurf1 controls osteoblast activity and bone homeostasis by targeting MEKK2 for degradation

Bone histomorphometry: standardization of nomenclature, symbols, and units. Report of the ASBMR Histomorphometry Nomenclature Committee

Effect of parathyroid hormone (1-34) on fractures and bone mineral density in postmenopausal women with osteoporosis

Smad signaling in skeletal development and regeneration.

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360-376

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scholarly article

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10.1111/ACEL.12566

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2

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