Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift. - Wikidata - thesis-toulmin - TriplyDB

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

http://www.wikidata.org/entity/Q37701122

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The angiotensin II type 2 receptor activates flow-mediated outward remodelling through T cells-dependent interleukin-17 production.

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Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

http://www.wikidata.org/entity/Q37701122

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on February 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@en

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@nl

type

label

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@en

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@nl

prefLabel

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@en

Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@nl

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CIRCRESAHA.114.303045

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Circulation Research

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Angiotensin II, from vasoconstrictor to growth factor: a paradigm shift.

@en

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Sasa Vukelic

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P2860

Angiotensin-converting enzyme inhibitors reduce mortality in hypertension: a meta-analysis of randomized clinical trials of renin-angiotensin-aldosterone system inhibitors involving 158,998 patients

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2013 ACCF/AHA guideline for the management of heart failure: a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines

Myocardin and Prx1 contribute to angiotensin II-induced expression of smooth muscle alpha-actin.

Control of SRF binding to CArG box chromatin regulates smooth muscle gene expression in vivo.

Mechanisms of angiotensin II- and arginine vasopressin-induced increases in protein synthesis and content in cultured rat aortic smooth muscle cells. Evidence for selective increases in smooth muscle isoactin expression.

Thrombin stimulates proliferation of cultured rat aortic smooth muscle cells by a proteolytically activated receptor

Angiotensin II induces hypertrophy, not hyperplasia, of cultured rat aortic smooth muscle cells.

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754-757

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scholarly article

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10.1161/CIRCRESAHA.114.303045

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5

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114

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Kathy K. Griendling

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2014-02-01T00:00:00Z

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260431044

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24577962

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