Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
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Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesisCross talk between EBV and telomerase: the role of TERT and NOTCH2 in the switch of latent/lytic cycle of the virusLong non-coding RNA H19 enhances cell proliferation and anchorage-independent growth of cervical cancer cell lines.Lenalidomide, Thalidomide, and Pomalidomide Reactivate the Epstein-Barr Virus Lytic Cycle through Phosphoinositide 3-Kinase Signaling and Ikaros Expression.Infection as a cause of childhood leukemia: virus detection employing whole genome sequencing.Interferon regulatory factor 8 regulates caspase-1 expression to facilitate Epstein-Barr virus reactivation in response to B cell receptor stimulation and chemical induction.
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Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 February 2014
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vedecký článok
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vetenskaplig artikel
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Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
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Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
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Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
@en
Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
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Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
@en
Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.
@nl
P2093
P2860
P50
P356
P1433
P1476
Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells
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P2093
Eric C Johannsen
Janet E Mertz
Shannon C Kenney
Sinisa Dovat
P2860
P304
P356
10.1128/JVI.03706-13
P407
P577
2014-02-12T00:00:00Z