Neonatal chlamydial infection induces mixed T-cell responses that drive allergic airway disease.
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Chlamydia pneumoniae-specific IgE is prevalent in asthma and is associated with disease severityElucidating novel disease mechanisms in severe asthmaPlatelet-activating factor receptor plays a role in lung injury and death caused by Influenza A in miceProtective immunity and safety of a genetically modified influenza virus vaccineNatural killer cells regulate Th1/Treg and Th17/Treg balance in chlamydial lung infectionHighly pathogenic avian influenza A H5N1 and pandemic H1N1 virus infections have different phenotypes in Toll-like receptor 3 knockout mice.Complement C5 activation during influenza A infection in mice contributes to neutrophil recruitment and lung injury.Infection-mediated asthma: etiology, mechanisms and treatment options, with focus on Chlamydia pneumoniae and macrolides.Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?Allergen-encoding bone marrow transfer inactivates allergic T cell responses, alleviating airway inflammation.A new short-term mouse model of chronic obstructive pulmonary disease identifies a role for mast cell tryptase in pathogenesis.Interleukin-13 promotes susceptibility to chlamydial infection of the respiratory and genital tracts.Haemophilus influenzae infection drives IL-17-mediated neutrophilic allergic airways diseaseImportance of mast cell Prss31/transmembrane tryptase/tryptase-γ in lung function and experimental chronic obstructive pulmonary disease and colitisTLR2, but not TLR4, is required for effective host defence against Chlamydia respiratory tract infection in early life.Chlamydia muridarum lung infection in infants alters hematopoietic cells to promote allergic airway disease in mice.Reduction of tumstatin in asthmatic airways contributes to angiogenesis, inflammation, and hyperresponsiveness.Evidence of infectious asthma phenotype: Chlamydia-induced allergy and pathogen-specific IgE in a neonatal mouse model.TLR2, TLR4 AND MyD88 Mediate Allergic Airway Disease (AAD) and Streptococcus pneumoniae-Induced Suppression of AAD.Schistosoma mansoni Tegument (Smteg) Induces IL-10 and Modulates Experimental Airway Inflammation.Severity of allergic airway disease due to house dust mite allergen is not increased after clinical recovery of lung infection with Chlamydia pneumoniae in mice.Update in asthma 2007.Programmed Death Ligand 1 Promotes Early-Life Chlamydia Respiratory Infection-Induced Severe Allergic Airway Disease.Pulmonary immunity during respiratory infections in early life and the development of severe asthma.Tumor necrosis factor-related apoptosis-inducing ligand translates neonatal respiratory infection into chronic lung disease.Constitutive production of IL-13 promotes early-life Chlamydia respiratory infection and allergic airway disease.Effect of neonatal respiratory infection on adult BALB/c hippocampal glucocorticoid and mineralocorticoid receptors.Neonatal chlamydial pneumonia induces altered respiratory structure and function lasting into adult life.Early life infection alters adult BALB/c hippocampal gene expression in a sex specific manner.Th2 cytokine antagonists: potential treatments for severe asthma.Neonatal immunology: responses to pathogenic microorganisms and epigenetics reveal an "immunodiverse" developmental state.Programming of the lung by early-life infection.Mouse models of severe asthma: Understanding the mechanisms of steroid resistance, tissue remodelling and disease exacerbation.Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma.LF-15 & T7, synthetic peptides derived from tumstatin, attenuate aspects of airway remodelling in a murine model of chronic OVA-induced allergic airway disease.Identification of IFN-γ and IL-27 as Critical Regulators of Respiratory Syncytial Virus-Induced Exacerbation of Allergic Airways Disease in a Mouse Model.Toll-like receptor 2 and 4 have Opposing Roles in the Pathogenesis of Cigarette Smoke-induced Chronic Obstructive Pulmonary Disease.Targeting PP2A and proteasome activity ameliorates features of allergic airway disease in mice.Phosphoinositide 3-kinase γ plays a critical role in bleomycin-induced pulmonary inflammation and fibrosis in mice.Respiratory Infection Induce Release of Hepoxilin A and Histamine Production by Airway Neutrophils
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Neonatal chlamydial infection induces mixed T-cell responses that drive allergic airway disease.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 June 2007
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Neonatal chlamydial infection ...... drive allergic airway disease.
@en
Neonatal chlamydial infection ...... drive allergic airway disease.
@nl
type
label
Neonatal chlamydial infection ...... drive allergic airway disease.
@en
Neonatal chlamydial infection ...... drive allergic airway disease.
@nl
prefLabel
Neonatal chlamydial infection ...... drive allergic airway disease.
@en
Neonatal chlamydial infection ...... drive allergic airway disease.
@nl
P2093
P2860
P50
P1476
Neonatal chlamydial infection ...... drive allergic airway disease
@en
P2093
Gerard E Kaiko
Jay C Horvat
Julie A Preston
Margaret A Wade
Nicole G Hansbro
Paul S Foster
Philip M Hansbro
P2860
P304
P356
10.1164/RCCM.200607-1005OC
P407
P577
2007-06-28T00:00:00Z