Diabetes, aging, and their tissue complications. - Wikidata - thesis-toulmin - TriplyDB

Diabetes, aging, and their tissue complications.

Diabetes, aging, and their tissue complications.

http://www.wikidata.org/entity/Q38208801

about

The Mediterranean Lifestyle as a Non-Pharmacological and Natural Antioxidant for Healthy Aging Consequences of metabolic and oxidative modifications of cartilage tissue Pathogenesis of diabetic cerebral vascular disease complication Microscopic observation of chemical modification in sections using scanning acoustic microscopy.Antiglycation and Antioxidant Properties of Momordica charantia Crosstalk between advanced glycation end products (AGEs)-receptor RAGE axis and dipeptidyl peptidase-4-incretin system in diabetic vascular complications.RAGE-aptamer Attenuates the Growth and Liver Metastasis of Malignant Melanoma in Nude Mice.Sevelamer reduces endothelial inflammatory response to advanced glycation end products.Advanced Glycation-Modified Human Serum Albumin Evokes Alterations in Membrane and Eryptosis in Erythrocytes.Aquaporin Membrane Channels in Oxidative Stress, Cell Signaling, and Aging: Recent Advances and Research Trends.Evaluation of aging, diabetes mellitus, and skin wounds by scanning acoustic microscopy with protease digestion Aqueous Extract of Glucoraphanin-Rich Broccoli Sprouts Inhibits Formation of Advanced Glycation End Products and Attenuates Inflammatory Reactions in Endothelial Cells

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Diabetes, aging, and their tissue complications.

http://www.wikidata.org/entity/Q38208801

description

article científic

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article scientifique

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Diabetes, aging, and their tissue complications.

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Diabetes, aging, and their tissue complications.

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Diabetes, aging, and their tissue complications.

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Journal of Clinical Investigation

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Diabetes, aging, and their tissue complications.

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Rick Bucala

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Tobacco smoke is a source of toxic reactive glycation products

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

Biochemistry and molecular cell biology of diabetic complications

An agent cleaving glucose-derived protein crosslinks in vitro and in vivo

Benfotiamine blocks three major pathways of hyperglycemic damage and prevents experimental diabetic retinopathy

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide

Advanced glycosylation products quench nitric oxide and mediate defective endothelium-dependent vasodilatation in experimental diabetes.

Aminoguanidine prevents diabetes-induced arterial wall protein cross-linking.

Advanced Maillard reaction end products are associated with Alzheimer disease pathology

Advanced glycosylation endproducts block the antiproliferative effect of nitric oxide. Role in the vascular and renal complications of diabetes mellitus.

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1887-1888

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scholarly article

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10.1172/JCI75224

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5

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124

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2014-05-01T00:00:00Z

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24789881

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4001559

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