about
STAT3-driven transcription depends upon the dimethylation of K49 by EZH2.Myeloid STAT3 promotes formation of colitis-associated colorectal cancer in miceScutellaria barbata D. Don inhibits growth and induces apoptosis by suppressing IL-6-inducible STAT3 pathway activation in human colorectal cancer cells.Lithocholic Acid Stimulates IL-8 Expression in Human Colorectal Cancer Cells Via Activation of Erk1/2 MAPK and Suppression of STAT3 Activity.Role of bile acids in colon carcinogenesis
P2860
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
STAT3: An Anti-Invasive Factor in Colorectal Cancer?
@en
type
label
STAT3: An Anti-Invasive Factor in Colorectal Cancer?
@en
prefLabel
STAT3: An Anti-Invasive Factor in Colorectal Cancer?
@en
P2093
P2860
P356
P1433
P1476
STAT3: An Anti-Invasive Factor in Colorectal Cancer?
@en
P2093
Alexandra R Harris
Jongdae Lee
P2860
P304
P356
10.3390/CANCERS6031394
P577
2014-07-03T00:00:00Z