Neutralization of IL-12 in vivo prevents induction of contact hypersensitivity and induces hapten-specific tolerance.
about
Proanthocyanidins inhibit UV-induced immunosuppression through IL-12-dependent stimulation of CD8+ effector T cells and inactivation of CD4+ T cells.Different role of IL-4 in the onset of hapten-induced contact hypersensitivity in BALB/c and C57BL/6 mice.CD8+ IL-17-producing T cells are important in effector functions for the elicitation of contact hypersensitivity responses.Activated complement component 3 (C3) is required for ultraviolet induction of immunosuppression and antigenic toleranceSkin-infiltrating monocytes/macrophages migrate to draining lymph nodes and produce IL-10 after contact sensitizer exposure to UV-irradiated skin.Cytip regulates dendritic-cell function in contact hypersensitivity.Leucocyte-associated immunoglobulin-like receptor-1 is an inhibitory regulator of contact hypersensitivity.
P2860
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P2860
Neutralization of IL-12 in vivo prevents induction of contact hypersensitivity and induces hapten-specific tolerance.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Neutralization of IL-12 in viv ...... ces hapten-specific tolerance.
@en
type
label
Neutralization of IL-12 in viv ...... ces hapten-specific tolerance.
@en
prefLabel
Neutralization of IL-12 in viv ...... ces hapten-specific tolerance.
@en
P2093
P1476
Neutralization of IL-12 in viv ...... uces hapten-specific tolerance
@en
P2093
Trinchieri G
P304
P407
P577
1996-03-01T00:00:00Z