Low-dose cadmium exposure induces peribronchiolar fibrosis through site-specific phosphorylation of vimentin.

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Low-dose cadmium exposure induces peribronchiolar fibrosis through site-specific phosphorylation of vimentin.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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Low-dose cadmium exposure indu ...... c phosphorylation of vimentin.

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Low-dose cadmium exposure indu ...... c phosphorylation of vimentin.

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Low-dose cadmium exposure indu ...... c phosphorylation of vimentin.

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P1476

Low-dose cadmium exposure indu ...... c phosphorylation of vimentin.

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P2093

Fu Jun Li

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Gang Liu

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Huashi Li

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Mohammad Athar

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Ranu Surolia

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Rui-Ming Liu

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Sergey B Mirov

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Veena B Antony

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Victor J Thannickal

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Zheng Wang

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P2860

Cellular and connective tissue changes in alveolar septal walls in emphysema

Yap1 acts downstream of α-catenin to control epidermal proliferation

Vimentin is a novel AKT1 target mediating motility and invasion

Stimulation of p38 mitogen-activated protein kinase is an early regulatory event for the cadmium-induced apoptosis in human promonocytic cells

Role of YAP/TAZ in mechanotransduction

The nature of small-airway obstruction in chronic obstructive pulmonary disease

The intermediate filament protein vimentin is a new target for epigallocatechin gallate.

Akt phosphorylates the Yes-associated protein, YAP, to induce interaction with 14-3-3 and attenuation of p73-mediated apoptosis.

The pathology of chronic obstructive pulmonary disease.

Yap1 is required for endothelial to mesenchymal transition of the atrioventricular cushion.

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scholarly article

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10.1152/AJPLUNG.00087.2017

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313

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2017-04-27T00:00:00Z

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28450285

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5538875

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