Exploiting the WNT Signaling Pathway for Clinical Purposes.

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Exploiting the WNT Signaling Pathway for Clinical Purposes.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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Exploiting the WNT Signaling Pathway for Clinical Purposes.

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Exploiting the WNT Signaling Pathway for Clinical Purposes.

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Item

label

Exploiting the WNT Signaling Pathway for Clinical Purposes.

@en

Exploiting the WNT Signaling Pathway for Clinical Purposes.

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Exploiting the WNT Signaling Pathway for Clinical Purposes.

@en

Exploiting the WNT Signaling Pathway for Clinical Purposes.

@nl

P1476

Exploiting the WNT Signaling Pathway for Clinical Purposes.

@en

P2093

Mark L Johnson

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Robert R Recker

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P2860

Osteocyte control of bone formation via sclerostin, a novel BMP antagonist

Sost down-regulation by mechanical strain in human osteoblastic cells involves PGE2 signaling via EP4

Bone dysplasia sclerosteosis results from loss of the SOST gene product, a novel cystine knot-containing protein

A mutation in the LDL receptor-related protein 5 gene results in the autosomal dominant high-bone-mass trait

Sclerostin is an osteocyte-expressed negative regulator of bone formation, but not a classical BMP antagonist

Linkage of a gene causing high bone mass to human chromosome 11 (11q12-13)

Identification of a 52 kb deletion downstream of the SOST gene in patients with van Buchem disease

TGF-β and BMP signaling in osteoblast, skeletal development, and bone formation, homeostasis and disease

Functional interaction between beta-catenin and FOXO in oxidative stress signaling

P2888

s11914-017-0357-0

P304

153-161

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P31

scholarly article

P356

10.1007/S11914-017-0357-0

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-04-21T00:00:00Z

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P698

28432596

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