Thalidomide ameliorates portal hypertension via nitric oxide synthase independent reduced systolic blood pressure.

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Novel treatment options for portal hypertension.Ultraviolet radiation, vitamin D and the development of obesity, metabolic syndrome and type-2 diabetes.

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Thalidomide ameliorates portal hypertension via nitric oxide synthase independent reduced systolic blood pressure.

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http://www.wikidata.org/entity/Q39540696

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Thalidomide ameliorates portal ...... duced systolic blood pressure.

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Thalidomide ameliorates portal ...... duced systolic blood pressure.

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Item

label

Thalidomide ameliorates portal ...... duced systolic blood pressure.

@en

Thalidomide ameliorates portal ...... duced systolic blood pressure.

@nl

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Thalidomide ameliorates portal ...... duced systolic blood pressure.

@en

Thalidomide ameliorates portal ...... duced systolic blood pressure.

@nl

P1476

Thalidomide ameliorates portal ...... duced systolic blood pressure.

@en

P2093

Mary A Maluccio

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Nicholas G Theodorakis

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Vyacheslav A Korshunov

http://www.w3.org/2001/XMLSchema#string

Yining N Wang

http://www.w3.org/2001/XMLSchema#string

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Clinical manifestations of portal hypertension

Inhibition of hepatic tumour necrosis factor-alpha attenuates the anandamide-induced vasoconstrictive response in cirrhotic rat livers

G-protein-coupled receptor kinase interactor-1 (GIT1) is a new endothelial nitric-oxide synthase (eNOS) interactor with functional effects on vascular homeostasis.

Paraquat poisoning induces TNF-α-dependent iNOS/NO mediated hyporesponsiveness of the aorta to vasoconstrictors in rats.

Cirrhosis and its complications: evidence based treatment.

Portal hypertension: cytokines and endothelins.

Mice lacking inducible nitric oxide synthase are not resistant to lipopolysaccharide-induced death

Endothelial nitric-oxide synthase (eNOS) is activated through G-protein-coupled receptor kinase-interacting protein 1 (GIT1) tyrosine phosphorylation and Src protein

Endothelial dysfunction in cirrhosis and portal hypertension.

A crucial role for GRK2 in regulation of endothelial cell nitric oxide synthase function in portal hypertension.

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4126-4135

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scholarly article

P356

10.3748/WJG.V21.I14.4126

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English

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http://www.w3.org/2001/XMLSchema#string

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Nicholas J Skill

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2015-04-01T00:00:00Z

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275217589

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25892862

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thalidomide

systole

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4394073

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Thalidomide ameliorates portal hypertension via nitric oxide synthase independent reduced systolic blood pressure.

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P2860

P2860

Thalidomide ameliorates portal hypertension via nitric oxide synthase independent reduced systolic blood pressure.

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