The IL-33 receptor (ST2) regulates early IL-13 production in fungus-induced allergic airway inflammation.
about
Type two innate lymphoid cells: the Janus cells in health and disease.Follicular helper T cells are responsible for IgE responses to Der p 1 following house dust mite sensitization in mice.Intestinal helminth infection induces highly functional resident memory CD4+ T cells in mice.Lung epithelium is the major source of IL-33 and is regulated by IL-33-dependent and IL-33-independent mechanisms in pulmonary cryptococcosis.Repeated Mouse Lung Exposures to Stachybotrys chartarum Shift Immune Response from Type 1 to Type 2.Lung epithelium: barrier immunity to inhaled fungi and driver of fungal-associated allergic asthma.Interactions between Innate Lymphoid Cells and Cells of the Innate and Adaptive Immune System.Cryptococcus-Epithelial Interactions.Bone marrow type 2 innate lymphoid cells: a local source of interleukin-5 in interleukin-33-driven eosinophilia.Identification of T helper (Th)1- and Th2-associated antigens of Cryptococcus neoformans in a murine model of pulmonary infection.
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The IL-33 receptor (ST2) regulates early IL-13 production in fungus-induced allergic airway inflammation.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
The IL-33 receptor (ST2) regul ...... allergic airway inflammation.
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type
label
The IL-33 receptor (ST2) regul ...... allergic airway inflammation.
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prefLabel
The IL-33 receptor (ST2) regul ...... allergic airway inflammation.
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The IL-33 receptor (ST2) regul ...... allergic airway inflammation.
@en
P2093
A Grahnert
F Brombacher
M Protschka
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P304
P356
10.1038/MI.2015.106
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2015-11-11T00:00:00Z