Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation.
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Lack of PINK1 alters glia innate immune responses and enhances inflammation-induced, nitric oxide-mediated neuron death.Ameliorating mitochondrial dysfunction restores carbon ion-induced cognitive deficits via co-activation of NRF2 and PINK1 signaling pathway.Cellular and Molecular Basis of Neurodegeneration in Parkinson Disease.The Therapeutic Potential of Metformin in Neurodegenerative Diseases
P2860
Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Progression of pathology in PI ...... changes to neuroinflammation.
@en
type
label
Progression of pathology in PI ...... changes to neuroinflammation.
@en
prefLabel
Progression of pathology in PI ...... changes to neuroinflammation.
@en
P2093
P2860
P50
P1476
Progression of pathology in PI ...... changes to neuroinflammation.
@en
P2093
Bastian Roller
Blas Morales-Gordo
Georg Auburger
Hans-Hermann Hoepken
Irmgard Tegeder
Júlia Canet-Pons
Lucie Valek
Michael Walter
Suzana Gispert
P2860
P2888
P356
10.1186/S12974-017-0928-0
P577
2017-08-02T00:00:00Z
P6179
1090954565