Increase in decorin and biglycan in Duchenne Muscular Dystrophy: role of fibroblasts as cell source of these proteoglycans in the disease.
about
Early transplantation of human immature dental pulp stem cells from baby teeth to golden retriever muscular dystrophy (GRMD) dogs: Local or systemic?Ahnak1 abnormally localizes in muscular dystrophies and contributes to muscle vesicle releaseInhibition of extracellular matrix assembly induces the expression of osteogenic markers in skeletal muscle cells by a BMP-2 independent mechanism.Perimysial fibroblasts of extraocular muscle, as unique as the muscle fibersExclusion of biglycan mutations in a cohort of patients with neuromuscular disorders.Collagen crosslinking does not dictate stiffness in a transgenic mouse model of skeletal muscle fibrosisDecorin interacts with connective tissue growth factor (CTGF)/CCN2 by LRR12 inhibiting its biological activityACE2 is augmented in dystrophic skeletal muscle and plays a role in decreasing associated fibrosis.High levels of the extracellular matrix proteoglycan decorin are associated with inhibition of testicular function.Biglycan: a multivalent proteoglycan providing structure and signalsImatinib attenuates skeletal muscle dystrophy in mdx miceConcurrent Label-Free Mass Spectrometric Analysis of Dystrophin Isoform Dp427 and the Myofibrosis Marker Collagen in Crude Extracts from mdx-4cv Skeletal Muscles.Structure and function of the skeletal muscle extracellular matrix.Cell-matrix interactions in muscle disease.Role of proteoglycans in the regulation of the skeletal muscle fibrotic response.Cardiac and respiratory dysfunction in Duchenne muscular dystrophy and the role of second messengers.Syndecans in skeletal muscle development, regeneration and homeostasis.Pathoproteomic profiling of the skeletal muscle matrisome in dystrophinopathy associated myofibrosis.Transforming growth factor type-β inhibits Mas receptor expression in fibroblasts but not in myoblasts or differentiated myotubes; Relevance to fibrosis associated to muscular dystrophies.The role of TGF-β1 during skeletal muscle regeneration.The emerging role of skeletal muscle extracellular matrix remodelling in obesity and exercise.The internal region leucine-rich repeat 6 of decorin interacts with low density lipoprotein receptor-related protein-1, modulates transforming growth factor (TGF)-β-dependent signaling, and inhibits TGF-β-dependent fibrotic response in skeletal muscCTGF/CCN-2 over-expression can directly induce features of skeletal muscle dystrophy.Angiotensin II receptor type 1 blockade decreases CTGF/CCN2-mediated damage and fibrosis in normal and dystrophic skeletal muscles.Constitutively activated dystrophic muscle fibroblasts show a paradoxical response to TGF-beta and CTGF/CCN2.Elastase levels and activity are increased in dystrophic muscle and impair myoblast cell survival, proliferation and differentiation.Immunoneutralization of TGFbeta1 Improves Skeletal Muscle Regeneration: Effects on Myoblast Differentiation and Glycosaminoglycan Content.Connective tissue cells expressing fibro/adipogenic progenitor markers increase under chronic damage: relevance in fibroblast-myofibroblast differentiation and skeletal muscle fibrosis.Corticosteroid effects on blood gene expression in Duchenne muscular dystrophy.Measurement of novel biomarkers to predict chronic heart failure outcomes and left ventricular remodeling.The pro-fibrotic connective tissue growth factor (CTGF/CCN2) correlates with the number of necrotic-regenerative foci in dystrophic muscle.Expression of CTGF/CCN2 in response to LPA is stimulated by fibrotic extracellular matrix via the integrin/FAK axis.Plasticity of the Muscle Stem Cell Microenvironment.
P2860
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P2860
Increase in decorin and biglycan in Duchenne Muscular Dystrophy: role of fibroblasts as cell source of these proteoglycans in the disease.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Increase in decorin and biglyc ...... proteoglycans in the disease.
@en
type
label
Increase in decorin and biglyc ...... proteoglycans in the disease.
@en
prefLabel
Increase in decorin and biglyc ...... proteoglycans in the disease.
@en
P2093
P2860
P1476
Increase in decorin and biglyc ...... proteoglycans in the disease.
@en
P2093
Daniel Cabrera
Jenny Holmgren
Karin Alvarez
Ricardo Fadic
Valeria Mezzano
P2860
P304
P356
10.1111/J.1582-4934.2006.TB00435.X
P577
2006-07-01T00:00:00Z