A cascade of morphogenic signaling initiated by the meninges controls corpus callosum formation.
about
Functional synergy between cholecystokinin receptors CCKAR and CCKBR in mammalian brain developmentAstroglial-mediated remodeling of the interhemispheric midline during telencephalic development is exclusive to eutherian mammalsEMX1 regulates NRP1-mediated wiring of the mouse anterior cingulate cortex.Polymicrogyria: pathology, fetal origins and mechanisms.Migration of oligodendrocyte progenitor cells is controlled by transforming growth factor β family proteins during corticogenesis.Deficiency of the chromatin regulator BRPF1 causes abnormal brain development.CoupTFI interacts with retinoic acid signaling during cortical development.Neural crest-derived mesenchymal cells require Wnt signaling for their development and drive invagination of the telencephalic midlineThalamic WNT3 Secretion Spatiotemporally Regulates the Neocortical Ribosome Signature and mRNA Translation to Specify Neocortical Cell Subtypes.Epithelial cells supply Sonic Hedgehog to the perinatal dentate gyrus via transport by plateletsBMP receptor 1A regulates development of hypothalamic circuits critical for feeding behavior.Bone morphogenic protein signaling is a major determinant of dentate development.Foxc1 is required by pericytes during fetal brain angiogenesis.Gli3 controls corpus callosum formation by positioning midline guideposts during telencephalic patterning.Astroglial-Mediated Remodeling of the Interhemispheric Midline Is Required for the Formation of the Corpus CallosumHeparan sulfotransferases Hs6st1 and Hs2st keep Erk in check for mouse corpus callosum development.Axon guidance mechanisms for establishment of callosal connections.Meninges: from protective membrane to stem cell niche.BMP signaling in telencephalic neural cell specification and maturation.Cerebrovascular defects in Foxc1 mutants correlate with aberrant WNT and VEGF-A pathways downstream of retinoic acid from the meninges.De novo inbred heterozygous Zeb2/Sip1 mutant mice uniquely generated by germ-line conditional knockout exhibit craniofacial, callosal and behavioral defects associated with Mowat-Wilson syndrome.Two specific populations of GABAergic neurons originating from the medial and the caudal ganglionic eminences aid in proper navigation of callosal axons.The evolution of cortical development: the synapsid-diapsid divergence.Meninges-derived cues control axon guidance.Increased FGF8 signaling promotes chondrogenic rather than osteogenic development in the embryonic skull.Building Spinal and Brain Commissures: Axon Guidance at the MidlineCongenital Hydrocephalus, Corpus Callosum Agenesis, and Prosencephalic Cyst with Supernumerary Nostril: A Neurocristopathy
P2860
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P2860
A cascade of morphogenic signaling initiated by the meninges controls corpus callosum formation.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
A cascade of morphogenic signa ...... ols corpus callosum formation.
@en
A cascade of morphogenic signa ...... ols corpus callosum formation.
@nl
type
label
A cascade of morphogenic signa ...... ols corpus callosum formation.
@en
A cascade of morphogenic signa ...... ols corpus callosum formation.
@nl
prefLabel
A cascade of morphogenic signa ...... ols corpus callosum formation.
@en
A cascade of morphogenic signa ...... ols corpus callosum formation.
@nl
P2093
P2860
P1433
P1476
A cascade of morphogenic signa ...... ols corpus callosum formation.
@en
P2093
Julie A Siegenthaler
Samuel J Pleasure
Youngshik Choe
P2860
P304
P356
10.1016/J.NEURON.2011.11.036
P407
P577
2012-02-01T00:00:00Z