Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
about
Roles and Modalities of Ectonucleotidases in Remodeling the Multiple Myeloma Niche.Inhibition of A2A Adenosine Receptor Signaling in Cancer Cells Proliferation by the Novel Antagonist TP455.New Strategies Using Antibody Combinations to Increase Cancer Treatment Effectiveness.Clinical significance of CD73 in triple-negative breast cancer: multiplex analysis of a phase III clinical trial.Extracellular 5'-nucleotidase (CD73) promotes human breast cancer cells growth through AKT/GSK-3β/β-catenin/cyclinD1 signaling pathway.Cytokine-Induced Killer Cells Express CD39, CD38, CD203a, CD73 Ectoenzymes and P1 Adenosinergic Receptors.Adenosinergic Signaling Alters Natural Killer Cell Functional Responses
P2860
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P2860
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@en
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@nl
type
label
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@en
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@nl
prefLabel
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@en
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@nl
P2093
P2860
P1433
P1476
Anti-CD73 immunotherapy: A viable way to reprogram the tumor microenvironment.
@en
P2093
Davide Ferrari
Fabio Malavasi
György Haskó
Luca Antonioli
P2860
P304
P356
10.1080/2162402X.2016.1216292
P577
2016-07-29T00:00:00Z