about
Advances in understanding chromosome silencing by the long non-coding RNA XistEssential role for de novo DNA methyltransferase Dnmt3a in paternal and maternal imprintingHistone variants enriched in oocytes enhance reprogramming to induced pluripotent stem cellsMITOPLD is a mitochondrial protein essential for nuage formation and piRNA biogenesis in the mouse germlineRelationship between DNA methylation, histone H4 acetylation and gene expression in the mouse imprinted Igf2-H19 domain.The CENP-O complex requirement varies among different cell types.Three novel DNMT3B mutations in Japanese patients with ICF syndrome.Role for piRNAs and noncoding RNA in de novo DNA methylation of the imprinted mouse Rasgrf1 locus.Species-specific differences in X chromosome inactivation in mammals.X chromosome reactivation dynamics reveal stages of reprogramming to pluripotencyA proximal conserved repeat in the Xist gene is essential as a genomic element for X-inactivation in mouse.Imprinted X inactivation and reprogramming in the preimplantation mouse embryo.Maintenance of Xist Imprinting Depends on Chromatin Condensation State and Rnf12 Dosage in Mice.An Xist-activating antisense RNA required for X-chromosome inactivation.Role of de novo DNA methyltransferases in initiation of genomic imprinting and X-chromosome inactivation.Targeted gene silencing in mouse germ cells by insertion of a homologous DNA into a piRNA generating locus.Large-scale identification and mapping of nuclear matrix-attachment regions in the distal imprinted domain of mouse chromosome 7.Effect of CpG methylation on expression of the mouse imprinted gene Mest.Epigenotype switching of imprintable loci in embryonic germ cells.Defects in dosage compensation impact global gene regulation in the mouse trophoblast.De novo DNA methylation independent establishment of maternal imprint on X chromosome in mouse oocytes.Tsix silences Xist through modification of chromatin structure.Control of Chromosomal Localization of Xist by hnRNP U Family Molecules.A new Xist allele driven by a constitutively active promoter is dominated by Xist locus environment and exhibits the parent-of-origin effects.Incomplete X-inactivation initiated by a hypomorphic Xist allele in the mouse.Sex-reversed somatic cell cloning in the mouse.Tsix defective in splicing is competent to establish Xist silencing.Tsix-deficient X chromosome does not undergo inactivation in the embryonic lineage in males: implications for Tsix-independent silencing of Xist.Crucial role of antisense transcription across the Xist promoter in Tsix-mediated Xist chromatin modification.De novo DNA methylation is dispensable for the initiation and propagation of X chromosome inactivation.Effect of TSIX disruption on XIST expression in male ES cells.What makes the maternal X chromosome resistant to undergoing imprinted X inactivation?Role of SmcHD1 in establishment of epigenetic states required for the maintenance of the X-inactivated state in miceHuman inactive X chromosome is compacted through a PRC2-independent SMCHD1-HBiX1 pathwayX Inactivation in the Mouse Embryo Deficient for Dnmt1: Distinct Effect of Hypomethylation on Imprinted and Random X InactivationPublisher Correction: Female mice lacking Ftx lncRNA exhibit impaired X-chromosome inactivation and a microphthalmia-like phenotypeFemale mice lacking Ftx lncRNA exhibit impaired X-chromosome inactivation and a microphthalmia-like phenotypeActivation of the inactive X chromosome induced by cell fusion between a murine EC and female somatic cell accompanies reproducible changes in the methylation pattern of the Xist geneImpeding Xist expression from the active X chromosome improves mouse somatic cell nuclear transfer
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description
hulumtues
@sq
researcher
@en
ricercatore
@it
wetenschapper
@nl
հետազոտող
@hy
name
Takashi Sado
@ast
Takashi Sado
@en
Takashi Sado
@es
Takashi Sado
@nl
Takashi Sado
@sl
type
label
Takashi Sado
@ast
Takashi Sado
@en
Takashi Sado
@es
Takashi Sado
@nl
Takashi Sado
@sl
prefLabel
Takashi Sado
@ast
Takashi Sado
@en
Takashi Sado
@es
Takashi Sado
@nl
Takashi Sado
@sl
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P1153
7006161014
P21
P214
2037154741635453110003
P31
P496
0000-0002-1232-0250
P569
2000-01-01T00:00:00Z
P735
P7859
lccn-n2019181059