N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

@zh

2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@en

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@nl

type

Item

label

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@en

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@nl

prefLabel

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@en

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@nl

P1476

N-acetylcysteine reverses cardiac myocyte dysfunction in HIV-Tat proteinopathy.

@en

P2093

Fangping Chen

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John M Hollander

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Mitchell S Finkel

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Walter Baseler

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William Lewis

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P2860

Appetizing rancidity of apoptotic cells for macrophages: oxidation, externalization, and recognition of phosphatidylserine

HIV-1 Tat Activates Neuronal Ryanodine Receptors with Rapid Induction of the Unfolded Protein Response and Mitochondrial Hyperpolarization

Molecular mechanism of decreased glutathione content in human immunodeficiency virus type 1 Tat-transgenic mice

Human immunodeficiency virus type 1 Tat protein impairs selenoglutathione peroxidase expression and activity by a mechanism independent of cellular selenium uptake: consequences on cellular resistance to UV-A radiation

HIV-1 viral proteins gp120 and Tat induce oxidative stress in brain endothelial cells

Cardiovascular manifestations in human immunodeficiency virus-infected patients.

Glutathione deficiency is associated with impaired survival in HIV disease.

Human immunodeficiency virus-1 Tat activates calpain proteases via the ryanodine receptor to enhance surface dopamine transporter levels and increase transporter-specific uptake and Vmax.

N-acetylcysteine prevents HIV gp 120-related damage of human cultured astrocytes: correlation with glutamine synthase dysfunction

Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, delta-sarcoglycan, in hamster: an animal model of disrupted dystrophin-associated glycoprotein complex

P304

105-113

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scholarly article

P356

10.1152/JAPPLPHYSIOL.00068.2012

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English

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P478

113

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P577

2012-05-03T00:00:00Z

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P5875

224897642

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22556393

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