Glycation of mitochondrial proteins from diabetic rat kidney is associated with excess superoxide formation.
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Glycation-altered proteolysis as a pathobiologic mechanism that links dietary glycemic index, aging, and age-related disease (in nondiabetics)Protein Modifications as Manifestations of Hyperglycemic Glucotoxicity in Diabetes and Its ComplicationsPharmacologic Approaches Against Advanced Glycation End Products (AGEs) in Diabetic Cardiovascular DiseaseEpigenetic Changes in Endothelial Progenitors as a Possible Cellular Basis for Glycemic Memory in Diabetic Vascular ComplicationsRole of methylglyoxal in Alzheimer's diseaseGeldanamycin derivative ameliorates high fat diet-induced renal failure in diabetesTargeting Mitochondria and Reactive Oxygen Species-Driven Pathogenesis in Diabetic NephropathyAlteration of renal respiratory Complex-III during experimental type-1 diabetesAcute hyperglycemia abolishes ischemic preconditioning by inhibiting Akt phosphorylation: normalizing blood glucose before ischemia restores ischemic preconditioning.Proteomics in renal research.Molecular mechanisms of diabetic vascular complicationsAminoguanidine inhibits aortic hydrogen peroxide production, VSMC NOX activity and hypercontractility in diabetic mice.Mitochondrial dysfunction in diabetes: from molecular mechanisms to functional significance and therapeutic opportunities.Hyperglycemia impairs proteasome function by methylglyoxalMethylglyoxal alters the function and stability of critical components of the protein quality control.Hyperglycaemia-induced impairment of endothelium-dependent vasorelaxation in rat mesenteric arteries is mediated by intracellular methylglyoxal levels in a pathway dependent on oxidative stressMinimal peroxide exposure of neuronal cells induces multifaceted adaptive responsesEffect of dietary prebiotic supplementation on advanced glycation, insulin resistance and inflammatory biomarkers in adults with pre-diabetes: a study protocol for a double-blind placebo-controlled randomised crossover clinical trial.Prevention of diabetic nephropathy in Ins2(+/)⁻(AkitaJ) mice by the mitochondria-targeted therapy MitoQ.Candesartan attenuates diabetic retinal vascular pathology by restoring glyoxalase-I function.Overexpression of glyoxalase-I reduces hyperglycemia-induced levels of advanced glycation end products and oxidative stress in diabetic ratsDoes chronic glycolysis accelerate aging? Could this explain how dietary restriction works?Impaired mitochondrial complex III and melatonin responsive reactive oxygen species generation in kidney mitochondria of db/db miceEarly- and advanced non-enzymatic glycation in diabetic vascular complications: the search for therapeutics.Role of advanced glycation endproducts and glyoxalase I in diabetic peripheral sensory neuropathy.Cumulative mtDNA damage and mutations contribute to the progressive loss of RGCs in a rat model of glaucomaPathological significance of mitochondrial glycationOxidation of fatty acids is the source of increased mitochondrial reactive oxygen species production in kidney cortical tubules in early diabetes.Mitochondrial glutathione transport: physiological, pathological and toxicological implications.Hypothesis: the 'metabolic memory', the new challenge of diabetes.A perspective on the Maillard reaction and the analysis of protein glycation by mass spectrometry: probing the pathogenesis of chronic diseaseProtein and nucleotide damage by glyoxal and methylglyoxal in physiological systems--role in ageing and disease.Multiple superoxide dismutase 1/splicing factor serine alanine 15 variants are associated with the development and progression of diabetic nephropathy: the Diabetes Control and Complications Trial/Epidemiology of Diabetes Interventions and ComplicatRAGE-induced cytosolic ROS promote mitochondrial superoxide generation in diabetes.Methylglyoxal, diabetes mellitus and diabetic complications.Free radical generation by methylglyoxal in tissues.Role of nitrosative stress in the pathogenesis of diabetic vascular dysfunction.Diabetes and Kidney Disease: Role of Oxidative Stress.Succination of thiol groups in adipose tissue proteins in diabetes: succination inhibits polymerization and secretion of adiponectinKnockdown of glyoxalase 1 mimics diabetic nephropathy in nondiabetic mice.
P2860
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P2860
Glycation of mitochondrial proteins from diabetic rat kidney is associated with excess superoxide formation.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Glycation of mitochondrial pro ...... h excess superoxide formation.
@en
Glycation of mitochondrial pro ...... h excess superoxide formation.
@nl
type
label
Glycation of mitochondrial pro ...... h excess superoxide formation.
@en
Glycation of mitochondrial pro ...... h excess superoxide formation.
@nl
prefLabel
Glycation of mitochondrial pro ...... h excess superoxide formation.
@en
Glycation of mitochondrial pro ...... h excess superoxide formation.
@nl
P2093
P2860
P1476
Glycation of mitochondrial pro ...... h excess superoxide formation.
@en
P2093
Aylin M Ozdemir
Mariana G Rosca
Michael Brownlee
Miriam F Weiss
Tiberiu G Mustata
Timothy S Kern
Vincent M Monnier
P2860
P304
P356
10.1152/AJPRENAL.00415.2004
P577
2005-04-05T00:00:00Z