Loss of selenoprotein N function causes disruption of muscle architecture in the zebrafish embryo.
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Increased muscle stress-sensitivity induced by selenoprotein N inactivation in mouse: a mammalian model for SEPN1-related myopathySwimming into prominence: the zebrafish as a valuable tool for studying human myopathies and muscular dystrophies.Loss of myotubularin function results in T-tubule disorganization in zebrafish and human myotubular myopathyDelta-Notch signalling in segmentationSelenoprotein N is required for ryanodine receptor calcium release channel activity in human and zebrafish muscleTopology and dynamics of the zebrafish segmentation clock core circuitSelenoprotein N deficiency in mice is associated with abnormal lung developmentSatellite cell loss and impaired muscle regeneration in selenoprotein N deficiencyMuscle degeneration and leukocyte infiltration caused by mutation of zebrafish Fad24Zebrafish MTMR14 is required for excitation-contraction coupling, developmental motor function and the regulation of autophagy.Selenoprotein N is dynamically expressed during mouse development and detected early in muscle precursors.Selenoproteins: molecular pathways and physiological roles.Regulation and function of selenoproteins in human diseaseToxicity assessments of near-infrared upconversion luminescent LaF3:Yb,Er in early development of zebrafish embryosMulti-minicore DiseaseGene expression of endoplasmic reticulum resident selenoproteins correlates with apoptosis in various muscles of se-deficient chicks.Congenital muscular dystrophy. Part II: a review of pathogenesis and therapeutic perspectives.Zebrafish in the sea of mineral (iron, zinc, and copper) metabolism.Selenoprotein N in skeletal muscle: from diseases to function.The cardiovascular triad of dysfunctional angiogenesis.The neuromuscular differential diagnosis of joint hypermobility.Endoplasmic reticulum-resident selenoproteins as regulators of calcium signaling and homeostasis.The neuroprotective functions of selenoprotein M and its role in cytosolic calcium regulation.Lack of Apobec2-related proteins causes a dystrophic muscle phenotype in zebrafish embryosRoles of selenoprotein antioxidant protection in zebrafish, Danio rerio, subjected to dietary oxidative stress.Ex vivo correction of selenoprotein N deficiency in rigid spine muscular dystrophy caused by a mutation in the selenocysteine codon.Dietary selenium regulation of transcript abundance of selenoprotein N and selenoprotein W in chicken muscle tissues.Ubiquitous expression of selenoprotein N transcripts in chicken tissues and early developmental expression pattern in skeletal muscles.Small molecule screen in embryonic zebrafish using modular variations to target segmentation.Analysis of the Interactions Between Thioredoxin and 20 Selenoproteins in Chicken.Synchrony dynamics during initiation, failure, and rescue of the segmentation clock.Segmentation of the zebrafish axial skeleton relies on notochord sheath cells and not on the segmentation clock.
P2860
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P2860
Loss of selenoprotein N function causes disruption of muscle architecture in the zebrafish embryo.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@en
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@nl
type
label
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@en
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@nl
prefLabel
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@en
Loss of selenoprotein N functi ...... cture in the zebrafish embryo.
@nl
P2093
P1476
Loss of selenoprotein N functi ...... ecture in the zebrafish embryo
@en
P2093
Alain Lescure
Bernard Thisse
Christine Thisse
Colette Hindelang
P304
P356
10.1016/J.YEXCR.2006.10.005
P407
P577
2006-10-13T00:00:00Z