about
Rare mutations provide unique insight into oncogenic potential of STAT transcription factors.A haunted beast: Targeting STAT5BN642H in T-Cell Neoplasia.STAT5 is essential for IL-7-mediated viability, growth, and proliferation of T-cell acute lymphoblastic leukemia cellsThe Cooperative Relationship between STAT5 and Reactive Oxygen Species in Leukemia: Mechanism and Therapeutic Potential
P2860
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年学术文章
@wuu
2017年学术文章
@zh
2017年学术文章
@zh-cn
2017年学术文章
@zh-hans
2017年学术文章
@zh-my
2017年学术文章
@zh-sg
2017年學術文章
@yue
2017年學術文章
@zh-hant
name
STAT5BN642H is a driver mutation for T cell neoplasia.
@en
STAT5BN642H is a driver mutation for T cell neoplasia.
@nl
type
label
STAT5BN642H is a driver mutation for T cell neoplasia.
@en
STAT5BN642H is a driver mutation for T cell neoplasia.
@nl
prefLabel
STAT5BN642H is a driver mutation for T cell neoplasia.
@en
STAT5BN642H is a driver mutation for T cell neoplasia.
@nl
P2093
P2860
P50
P356
P1476
STAT5BN642H is a driver mutation for T cell neoplasia.
@en
P2093
Auke Boersma
Barbara Maurer
Eva Grundschober
Ha Thi Thanh Pham
Jan Pencik
Markus Hengstschläger
Matthias Farlik
Michaela Prchal-Murphy
Mohamed Elabd
Reinhard Grausenburger
P2860
P304
P356
10.1172/JCI94509
P407
P50
P577
2017-12-04T00:00:00Z