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Chronic intermittent hypoxia alters hypothalamic transcription of genes involved in metabolic regulationLingual muscle activity across sleep-wake States in rats with surgically altered upper airway.Chronic intermittent hypoxia alters density of aminergic terminals and receptors in the hypoglossal motor nucleus.Evidence that adrenergic ventrolateral medullary cells are activated whereas precerebellar lateral reticular nucleus neurons are suppressed during REM sleep.Antagonism of orexin 1 receptors eliminates motor hyperactivity and improves homing response acquisition in juvenile rats exposed to alcohol during early postnatal period.Rats subjected to chronic-intermittent hypoxia have increased density of noradrenergic terminals in the trigeminal sensory and motor nuclei.Regionally selective effects of GABA on hypothalamic GABAA receptor mRNA in vitro.Mesopontine cholinergic projections to the hypoglossal motor nucleus.Sleep-wake control of the upper airway by noradrenergic neurons, with and without intermittent hypoxia.REM sleep-like episodes of motoneuronal depression and respiratory rate increase are triggered by pontine carbachol microinjections in in situ perfused rat brainstem preparation.Central pathways of pulmonary and lower airway vagal afferents.Glucoregulatory consequences and cardiorespiratory parameters in rats exposed to chronic-intermittent hypoxia: effects of the duration of exposure and losartan.Pontine cholinergic mechanisms and their impact on respiratory regulation.Effect of chronic intermittent hypoxia on noradrenergic activation of hypoglossal motoneuronsInhibition of A5 Neurons Facilitates the Occurrence of REM Sleep-Like Episodes in Urethane-Anesthetized Rats: A New Role for Noradrenergic A5 Neurons?Reduced sleep and impaired sleep initiation in adult male rats exposed to alcohol during early postnatal period.Antagonism of orexin receptors in the posterior hypothalamus reduces hypoglossal and cardiorespiratory excitation from the perifornical hypothalamus.Reduced c-Fos expression in medullary catecholaminergic neurons in rats 20 h after exposure to chronic intermittent hypoxia.Adventures and tribulations in the search for the mechanisms of the atonia of REM sleep.Hypoglossal premotor neurons of the intermediate medullary reticular region express cholinergic markers.Developmental profiles of neurotransmitter receptors in respiratory motor nuclei.Circadian dependence of receptors that mediate wake-related excitatory drive to hypoglossal motoneurons.Respiratory modulation of lingual muscle activity across sleep-wake states in rats.Loss of motoneurons in the ventral compartment of the rat hypoglossal nucleus following early postnatal exposure to alcohol.Electromyographic activity at the base and tip of the tongue across sleep-wake states in rats.Antagonism of alpha1-adrenergic and serotonergic receptors in the hypoglossal motor nucleus does not prevent motoneuronal activation elicited from the posterior hypothalamus.Dorsomedial pontine neurons with descending projections to the medullary reticular formation express orexin-1 and adrenergic alpha2A receptor mRNATime- and behavioral state-dependent changes in posterior hypothalamic GABAA receptors contribute to the regulation of sleep.Differential localization of carbachol- and bicuculline-sensitive pontine sites for eliciting REM sleep-like effects in anesthetized rats.Neural Control of the Upper Airway: Respiratory and State-Dependent Mechanisms.The α1 adrenoceptor antagonist prazosin enhances sleep continuity in fear-conditioned Wistar-Kyoto rats.REM sleep-like atonia of hypoglossal (XII) motoneurons is caused by loss of noradrenergic and serotonergic inputs.Sleep and breathing. Foreword.A5 cells are silenced when REM sleep-like signs are elicited by pontine carbachol.Postnatal development of serotonin 1B, 2 A and 2C receptors in brainstem motoneurons.Activation of HIF-1alpha mRNA by hypoxia and iron chelator in isolated rat carotid body.Carbachol injections into the ventral pontine reticular formation activate locus coeruleus cells in urethane-anesthetized rats.Noradrenergic, serotonergic and GABAergic antagonists injected together into the XII nucleus abolish the REM sleep-like depression of hypoglossal motoneuronal activity.Single-cell RT-PCR gene expression profiling of acutely dissociated and immunocytochemically identified central neurons.Hypoglossal motoneurons are endogenously activated by serotonin during the active period of circadian cycle.
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description
hulumtues
@sq
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researcher
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հետազոտող
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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Leszek Kubin
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prefLabel
Leszek Kubin
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Leszek Kubin
@da
Leszek Kubin
@de
Leszek Kubin
@en
Leszek Kubin
@es
Leszek Kubin
@fo
Leszek Kubin
@fr
Leszek Kubin
@is
Leszek Kubin
@kl
Leszek Kubin
@nb