Nitric oxide signalling is required for the generation of anoxia-induced long-term potentiation in the hippocampus.
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The Effects of Hypoxia and Inflammation on Synaptic Signaling in the CNSProtein tyrosine kinase is required for the induction of long-term potentiation in the rat hippocampus.Ischemia induces short- and long-term remodeling of synaptic activity in the hippocampus.Global cerebral ischemia: synaptic and cognitive dysfunction.Exogenous nitric oxide causes potentiation of hippocampal synaptic transmission during low-frequency stimulation via the endogenous nitric oxide-cGMP pathway.Changes in the calcium dependence of glutamate transmission in the hippocampal CA1 region after brief hypoxia-hypoglycemia.Prior short-term synaptic disinhibition facilitates long-term potentiation and suppresses long-term depression at CA1 hippocampal synapses.
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P2860
Nitric oxide signalling is required for the generation of anoxia-induced long-term potentiation in the hippocampus.
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1997 nî lūn-bûn
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1997年の論文
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1997年学术文章
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1997年学术文章
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Nitric oxide signalling is req ...... tentiation in the hippocampus.
@en
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@nl
type
label
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@en
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@nl
prefLabel
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@en
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@nl
P2860
P1476
Nitric oxide signalling is req ...... tentiation in the hippocampus.
@en
P2093
P2860
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P356
10.1111/J.1460-9568.1997.TB01387.X
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P577
1997-10-01T00:00:00Z