about
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The tumor-suppressor function of E-cadherinQuantitative comparison of constitutive promoters in human ES cellsN-CAM exhibits a regulatory function in pathological angiogenesis in oxygen induced retinopathyA causal role for E-cadherin in the transition from adenoma to carcinomaCdc42/N-WASP signaling links actin dynamics to pancreatic β cell delamination and differentiation.Artificial three-dimensional niches deconstruct pancreas development in vitro.Cdc42-mediated tubulogenesis controls cell specification.Definitive endoderm: a key step in coaxing human embryonic stem cells into transplantable beta-cells.The multifunctional FUS, EWS and TAF15 proto-oncoproteins show cell type-specific expression patterns and involvement in cell spreading and stress response.Rac1 regulates pancreatic islet morphogenesisFGF4 and retinoic acid direct differentiation of hESCs into PDX1-expressing foregut endoderm in a time- and concentration-dependent manner.Cadherins in development.NANOG reporter cell lines generated by gene targeting in human embryonic stem cells.Growth-limiting role of endothelial cells in endoderm development.N-cadherin is dispensable for pancreas development but required for beta-cell granule turnover.Mutation of conserved cysteines in the Ly6 domain of GPIHBP1 in familial chylomicronemia.Distinct gene expression signatures in human embryonic stem cells differentiated towards definitive endoderm at single-cell level.Human embryonic stem cells: origin, properties and applications.Characterization of human embryonic stem cell lines by the International Stem Cell Initiative.Elimination of damaged proteins during differentiation of embryonic stem cellsEfficient Generation of Glucose-Responsive Beta Cells from Isolated GP2+ Human Pancreatic Progenitors.Pericytes: gatekeepers in tumour cell metastasis?Impaired insulin exocytosis in neural cell adhesion molecule-/- mice due to defective reorganization of the submembrane F-actin networkβ-Catenin Regulates Primitive Streak Induction through Collaborative Interactions with SMAD2/SMAD3 and OCT4.Removal of damaged proteins during ES cell fate specification requires the proteasome activator PA28Genetic dissection of cadherin function during nephrogenesis.The establishment of 20 different human embryonic stem cell lines and subclones; a report on derivation, culture, characterisation and banking.Hepatocyte-like cells derived from human embryonic stem cells specifically via definitive endoderm and a progenitor stage.FGF2 specifies hESC-derived definitive endoderm into foregut/midgut cell lineages in a concentration-dependent manner.Quantitative transcription factor analysis of undifferentiated single human embryonic stem cells.Derivation of a xeno-free human embryonic stem cell line.Clonal derivation and characterization of human embryonic stem cell lines.The International Stem Cell Initiative: toward benchmarks for human embryonic stem cell research.Human blastocysts for the development of embryonic stem cells.Derivation, characterization, and differentiation of human embryonic stem cells.Properties of the reverse transcription reaction in mRNA quantification.Reconstructing human pancreatic differentiation by mapping specific cell populations during development.Pericytes limit tumor cell metastasis.Artifactual insulin release from differentiated embryonic stem cells.Vascular function and sphingosine-1-phosphate regulate development of the dorsal pancreatic mesenchyme.
P50
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P50
description
onderzoeker
@nl
stem cell researcher at University of Copenhagen
@en
name
Henrik Semb
@ast
Henrik Semb
@da
Henrik Semb
@de
Henrik Semb
@en
Henrik Semb
@es
Henrik Semb
@fo
Henrik Semb
@fr
Henrik Semb
@is
Henrik Semb
@kl
Henrik Semb
@nb
type
label
Henrik Semb
@ast
Henrik Semb
@da
Henrik Semb
@de
Henrik Semb
@en
Henrik Semb
@es
Henrik Semb
@fo
Henrik Semb
@fr
Henrik Semb
@is
Henrik Semb
@kl
Henrik Semb
@nb
prefLabel
Henrik Semb
@ast
Henrik Semb
@da
Henrik Semb
@de
Henrik Semb
@en
Henrik Semb
@es
Henrik Semb
@fo
Henrik Semb
@fr
Henrik Semb
@is
Henrik Semb
@kl
Henrik Semb
@nb
P106
P21
P31
P496
0000-0002-6747-787X